Papers associated with nerve (and abcc8)

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	Mutations of the same conserved glutamate residue in NBD2 of the sulfonylurea receptor 1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes., Männikkö R., Diabetes. June 1, 2011; 60 (6): 1813-22.
	Activation of the K(ATP) channel by Mg-nucleotide interaction with SUR1., Proks P., J Gen Physiol. October 1, 2010; 136 (4): 389-405.
	Xenopus pancreas development., Pearl EJ., Dev Dyn. June 1, 2009; 238 (6): 1271-86.
	Functional effects of mutations at F35 in the NH2-terminus of Kir6.2 (KCNJ11), causing neonatal diabetes, and response to sulfonylurea therapy., Proks P., Diabetes. June 1, 2006; 55 (6): 1731-7.
	Functional effects of naturally occurring KCNJ11 mutations causing neonatal diabetes on cloned cardiac KATP channels., Tammaro P., J Physiol. February 15, 2006; 571 (Pt 1): 3-14.
	Functional effects of KCNJ11 mutations causing neonatal diabetes: enhanced activation by MgATP., Proks P., Hum Mol Genet. September 15, 2005; 14 (18): 2717-26.
	Ligand-dependent linkage of the ATP site to inhibition gate closure in the KATP channel., Li L., J Gen Physiol. September 1, 2005; 126 (3): 285-99.
	Relapsing diabetes can result from moderately activating mutations in KCNJ11., Gloyn AL., Hum Mol Genet. April 1, 2005; 14 (7): 925-34.
	Effect of two amino acids in TM17 of Sulfonylurea receptor SUR1 on the binding of ATP-sensitive K+ channel modulators., Hambrock A., Diabetes. December 1, 2004; 53 Suppl 3 S128-34.
	Identification of a functionally important negatively charged residue within the second catalytic site of the SUR1 nucleotide-binding domains., Campbell JD., Diabetes. December 1, 2004; 53 Suppl 3 S123-7.
	Taurine block of cloned ATP-sensitive K+ channels with different sulfonylurea receptor subunits expressed in Xenopus laevis oocytes., Lim JG., Biochem Pharmacol. September 1, 2004; 68 (5): 901-10.
	Zinc is both an intracellular and extracellular regulator of KATP channel function., Prost AL., J Physiol. August 15, 2004; 559 (Pt 1): 157-67.
	Mapping the architecture of the ATP-binding site of the KATP channel subunit Kir6.2., Dabrowski M., J Physiol. June 1, 2004; 557 (Pt 2): 347-54.
	Inhibition of ATP-sensitive K+ channels by taurine through a benzamido-binding site on sulfonylurea receptor 1., Park EJ., Biochem Pharmacol. March 15, 2004; 67 (6): 1089-96.
	Molecular analysis of the subtype-selective inhibition of cloned KATP channels by PNU-37883A., Kovalev H., Br J Pharmacol. March 1, 2004; 141 (5): 867-73.
	3-trifluoromethyl-4-nitro-5-arylpyrazoles are novel K(ATP) channel agonists., Peat AJ., Bioorg Med Chem Lett. February 9, 2004; 14 (3): 813-6.
	Potent and selective activation of the pancreatic beta-cell type K(ATP) channel by two novel diazoxide analogues., Dabrowski M., Diabetologia. October 1, 2003; 46 (10): 1375-82.
	Inhibition of ATP-sensitive K+ channels by substituted benzo[c]quinolizinium CFTR activators., Prost A., Biochem Pharmacol. August 1, 2003; 66 (3): 425-30.
	Pyridine nucleotide regulation of the KATP channel Kir6.2/SUR1 expressed in Xenopus oocytes., Dabrowski M., J Physiol. July 15, 2003; 550 (Pt 2): 357-63.
	Characterisation of new KATP-channel mutations associated with congenital hyperinsulinism in the Finnish population., Reimann F., Diabetologia. February 1, 2003; 46 (2): 241-9.
	K(ATP) channel activity is required for hatching in Xenopus embryos., Cheng SM., Dev Dyn. December 1, 2002; 225 (4): 588-91.
	Protons activate homomeric Kir6.2 channels by selective suppression of the long and intermediate closures., Wu J., J Membr Biol. November 15, 2002; 190 (2): 105-16.

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