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Different thresholds of Wnt- Frizzled 7 signaling coordinate proliferation, morphogenesis and fate of endoderm progenitor cells. , Zhang Z ., Dev Biol. June 1, 2013; 378 (1): 1-12.
Inhibition of FGF signaling converts dorsal mesoderm to ventral mesoderm in early Xenopus embryos. , Lee SY., Differentiation. September 1, 2011; 82 (2): 99-107.
ADMP2 is essential for primitive blood and heart development in Xenopus. , Kumano G ., Dev Biol. November 15, 2006; 299 (2): 411-23.
XBP1 forms a regulatory loop with BMP-4 and suppresses mesodermal and neural differentiation in Xenopus embryos. , Cao Y , Cao Y ., Mech Dev. January 1, 2006; 123 (1): 84-96.
Neural induction in Xenopus: requirement for ectodermal and endomesodermal signals via Chordin, Noggin, beta-Catenin, and Cerberus. , Kuroda H ., PLoS Biol. May 1, 2004; 2 (5): E92.
Cell-autonomous and signal-dependent expression of liver and intestine marker genes in pluripotent precursor cells from Xenopus embryos. , Chen Y , Chen Y ., Mech Dev. March 1, 2003; 120 (3): 277-88.
A role for GATA5 in Xenopus endoderm specification. , Weber H., Development. October 1, 2000; 127 (20): 4345-60.
Neuralization of the Xenopus embryo by inhibition of p300/ CREB-binding protein function. , Kato Y ., J Neurosci. November 1, 1999; 19 (21): 9364-73.
Cloning of Mix-related homeodomain proteins using fast retrieval of gel shift activities, (FROGS), a technique for the isolation of DNA-binding proteins. , Mead PE ., Proc Natl Acad Sci U S A. September 15, 1998; 95 (19): 11251-6.
Smad7 inhibits mesoderm formation and promotes neural cell fate in Xenopus embryos. , Bhushan A ., Dev Biol. August 15, 1998; 200 (2): 260-8.
The Xenopus dorsalizing factor Gremlin identifies a novel family of secreted proteins that antagonize BMP activities. , Hsu DR., Mol Cell. April 1, 1998; 1 (5): 673-83.
The ALK-2 and ALK-4 activin receptors transduce distinct mesoderm-inducing signals during early Xenopus development but do not co-operate to establish thresholds. , Armes NA., Development. October 1, 1997; 124 (19): 3797-804.