Papers associated with neuron (and nav1)

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	Characterization of Na+ currents regulating intrinsic excitability of optic tectal neurons., Thompson AC., Life Sci Alliance. January 1, 2024; 7 (1):
	Heterologous functional expression of ascidian Nav1 channels and close relationship with the evolutionary ancestor of vertebrate Nav channels., Kawai T., J Biol Chem. January 1, 2021; 296 100783.
	Chemical modification of proteins by insertion of synthetic peptides using tandem protein trans-splicing., Khoo KK., Nat Commun. May 8, 2020; 11 (1): 2284.
	The mechanosensitive ion channel TRAAK is localized to the mammalian node of Ranvier., Brohawn SG., Elife. November 1, 2019; 8
	Prdm12 Directs Nociceptive Sensory Neuron Development by Regulating the Expression of the NGF Receptor TrkA., Desiderio S., Cell Rep. March 26, 2019; 26 (13): 3522-3536.e5.
	Amyloid precursor protein modulates Nav1.6 sodium channel currents through a Go-coupled JNK pathway., Li S., Sci Rep. December 23, 2016; 6 39320.
	Mutant SOD1 protein increases Nav1.3 channel excitability., Kubat Öktem E., J Biol Phys. June 1, 2016; 42 (3): 351-70.
	Spatiotemporal integration of developmental cues in neural development., Borodinsky LN., Dev Neurobiol. April 1, 2015; 75 (4): 349-59.
	μ-Conotoxins that differentially block sodium channels NaV1.1 through 1.8 identify those responsible for action potentials in sciatic nerve., Wilson MJ., Proc Natl Acad Sci U S A. June 21, 2011; 108 (25): 10302-7.
	Induction of vertebrate regeneration by a transient sodium current., Tseng AS., J Neurosci. September 29, 2010; 30 (39): 13192-200.
	Effects of BmK AS on Nav1.2 expressed in Xenopus laevis oocytes., Tan M., Cell Biol Toxicol. April 1, 2008; 24 (2): 143-9.
	H+ pump-dependent changes in membrane voltage are an early mechanism necessary and sufficient to induce Xenopus tail regeneration., Adams DS., Development. April 1, 2007; 134 (7): 1323-35.
	Isolation and structure-activity of mu-conotoxin TIIIA, a potent inhibitor of tetrodotoxin-sensitive voltage-gated sodium channels., Lewis RJ., Mol Pharmacol. March 1, 2007; 71 (3): 676-85.
	muO-conotoxin MrVIB selectively blocks Nav1.8 sensory neuron specific sodium channels and chronic pain behavior without motor deficits., Ekberg J., Proc Natl Acad Sci U S A. November 7, 2006; 103 (45): 17030-5.
	Repressor element-1 silencing transcription/neuron-restrictive silencer factor is required for neural sodium channel expression during development of Xenopus., Armisén R., J Neurosci. October 1, 2002; 22 (19): 8347-51.

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