Papers associated with tail (and kcne1)

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	Functional Characterization of a Spectrum of Novel Romano-Ward Syndrome KCNQ1 Variants., Rinné S., Int J Mol Sci. January 10, 2023; 24 (2):
	Optimized tight binding between the S1 segment and KCNE3 is required for the constitutively open nature of the KCNQ1-KCNE3 channel complex., Kasuya G., Elife. November 4, 2022; 11
	Pharmacological rescue of specific long QT variants of KCNQ1/KCNE1 channels., Zou X., Front Physiol. January 1, 2022; 13 902224.
	Molecular Mechanism of Autosomal Recessive Long QT-Syndrome 1 without Deafness., Oertli A., Int J Mol Sci. January 23, 2021; 22 (3):
	Polyunsaturated fatty acid analogues differentially affect cardiac NaV, CaV, and KV channels through unique mechanisms., Bohannon BM., Elife. March 24, 2020; 9
	Structure and physiological function of the human KCNQ1 channel voltage sensor intermediate state., Taylor KC., Elife. February 25, 2020; 9
	ML277 specifically enhances the fully activated open state of KCNQ1 by modulating VSD-pore coupling., Hou P., Elife. July 22, 2019; 8
	Deconstruction of an African folk medicine uncovers a novel molecular strategy for therapeutic potassium channel activation., De Silva AM., Sci Adv. November 14, 2018; 4 (11): eaav0824.
	KCNE1 tunes the sensitivity of KV7.1 to polyunsaturated fatty acids by moving turret residues close to the binding site., Larsson JE., Elife. July 17, 2018; 7
	Direct neurotransmitter activation of voltage-gated potassium channels., Manville RW., Nat Commun. May 10, 2018; 9 (1): 1847.
	KCNE1 induces fenestration in the Kv7.1/KCNE1 channel complex that allows for highly specific pharmacological targeting., Wrobel E., Nat Commun. October 12, 2016; 7 12795.
	Polyunsaturated fatty acid analogs act antiarrhythmically on the cardiac IKs channel., Liin SI., Proc Natl Acad Sci U S A. May 5, 2015; 112 (18): 5714-9.
	High incidence of functional ion-channel abnormalities in a consecutive Long QT cohort with novel missense genetic variants of unknown significance., Steffensen AB., Sci Rep. January 12, 2015; 5 10009.
	Ginseng gintonin activates the human cardiac delayed rectifier K+ channel: involvement of Ca2+/calmodulin binding sites., Choi SH., Mol Cells. September 1, 2014; 37 (9): 656-63.
	Domain-domain interactions determine the gating, permeation, pharmacology, and subunit modulation of the IKs ion channel., Zaydman MA., Elife. March 12, 2014; 3 e03606.
	Differential effects of ginsenoside metabolites on slowly activating delayed rectifier K(+) and KCNQ1 K(+) channel currents., Choi SH., J Ginseng Res. July 1, 2013; 37 (3): 324-31.
	Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1., Aidery P., Gene. December 10, 2012; 511 (1): 26-33.
	KCNQ1 subdomains involved in KCNE modulation revealed by an invertebrate KCNQ1 orthologue., Nakajo K., J Gen Physiol. November 1, 2011; 138 (5): 521-35.
	Extracellular potassium inhibits Kv7.1 potassium channels by stabilizing an inactivated state., Larsen AP., Biophys J. August 17, 2011; 101 (4): 818-27.
	Transmembrane potential of GlyCl-expressing instructor cells induces a neoplastic-like conversion of melanocytes via a serotonergic pathway., Blackiston D., Dis Model Mech. January 1, 2011; 4 (1): 67-85.
	KCNE1 and KCNE3 beta-subunits regulate membrane surface expression of Kv12.2 K(+) channels in vitro and form a tripartite complex in vivo., Clancy SM., PLoS One. July 22, 2009; 4 (7): e6330.
	Discovery of a novel activator of KCNQ1-KCNE1 K channel complexes., Mruk K., PLoS One. January 1, 2009; 4 (1): e4236.
	Modulation of potassium channel function confers a hyperproliferative invasive phenotype on embryonic stem cells., Morokuma J., Proc Natl Acad Sci U S A. October 28, 2008; 105 (43): 16608-13.
	KCNE1 constrains the voltage sensor of Kv7.1 K+ channels., Shamgar L., PLoS One. April 9, 2008; 3 (4): e1943.
	KCNE peptides differently affect voltage sensor equilibrium and equilibration rates in KCNQ1 K+ channels., Rocheleau JM., J Gen Physiol. January 1, 2008; 131 (1): 59-68.
	The role of S4 charges in voltage-dependent and voltage-independent KCNQ1 potassium channel complexes., Panaghie G., J Gen Physiol. February 1, 2007; 129 (2): 121-33.
	Secondary structure of a KCNE cytoplasmic domain., Rocheleau JM., J Gen Physiol. December 1, 2006; 128 (6): 721-9.
	KCNE3 truncation mutants reveal a bipartite modulation of KCNQ1 K+ channels., Gage SD., J Gen Physiol. December 1, 2004; 124 (6): 759-71.
	Ranolazine: ion-channel-blocking actions and in vivo electrophysiological effects., Schram G., Br J Pharmacol. August 1, 2004; 142 (8): 1300-8.
	Acute effects of dronedarone on both components of the cardiac delayed rectifier K+ current, HERG and KvLQT1/minK potassium channels., Thomas D., Br J Pharmacol. November 1, 2003; 140 (5): 996-1002.
	KCNE2 modulates current amplitudes and activation kinetics of HCN4: influence of KCNE family members on HCN4 currents., Decher N., Pflugers Arch. September 1, 2003; 446 (6): 633-40.
	Clinical and electrophysiological characterization of a novel mutation (F193L) in the KCNQ1 gene associated with long QT syndrome., Yamaguchi M., Clin Sci (Lond). April 1, 2003; 104 (4): 377-82.
	Inhibitory effects of volatile anesthetics on currents produced on heterologous expression of KvLQT1 and minK in Xenopus oocytes., Chen X., Vascul Pharmacol. July 1, 2002; 39 (1-2): 33-8.
	The oxidant thimerosal modulates gating behavior of KCNQ1 by interaction with the channel outer shell., Kerst G., J Membr Biol. March 15, 2002; 186 (2): 89-100.
	Blocking effects of the antiarrhythmic drug propafenone on the HERG potassium channel., Mergenthaler J., Naunyn Schmiedebergs Arch Pharmacol. April 1, 2001; 363 (4): 472-80.
	MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
	Gating and flickery block differentially affected by rubidium in homomeric KCNQ1 and heteromeric KCNQ1/KCNE1 potassium channels., Pusch M., Biophys J. January 1, 2000; 78 (1): 211-26.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
	Increase of the single-channel conductance of KvLQT1 potassium channels induced by the association with minK., Pusch M., Pflugers Arch. December 1, 1998; 437 (1): 172-4.
	Activation and inactivation of homomeric KvLQT1 potassium channels., Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.
	Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits., Tristani-Firouzi M., J Physiol. July 1, 1998; 510 ( Pt 1) 37-45.
	Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.
	Block by propofol and thiopentone of the min K current (IsK) expressed in Xenopus oocytes., Heath BM., Naunyn Schmiedebergs Arch Pharmacol. September 1, 1997; 356 (3): 404-9.
	Mechanism of enhancement of slow delayed rectifier current by extracellular sulfhydryl modification., Yao JA., Am J Physiol. July 1, 1997; 273 (1 Pt 2): H208-19.
	Positive regulation by chloride channel blockers of IsK channels expressed in Xenopus oocytes., Busch AE., Mol Pharmacol. October 1, 1994; 46 (4): 750-3.
	Species variants of the IsK protein: differences in kinetics, voltage dependence, and La3+ block of the currents expressed in Xenopus oocytes., Hice RE., Pflugers Arch. January 1, 1994; 426 (1-2): 139-45.
	Effects of [Ca2+]i and temperature on minK channels expressed in Xenopus oocytes., Busch AE., FEBS Lett. November 15, 1993; 334 (2): 221-4.

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