Papers associated with nerve (and nav1)

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	The mechanosensitive ion channel TRAAK is localized to the mammalian node of Ranvier., Brohawn SG., Elife. November 1, 2019; 8
	Amyloid precursor protein modulates Nav1.6 sodium channel currents through a Go-coupled JNK pathway., Li S., Sci Rep. December 23, 2016; 6 39320.
	Bioelectric memory: modeling resting potential bistability in amphibian embryos and mammalian cells., Law R., Plant Physiol. October 15, 2015; 12 22.
	A gamut of undiscovered electrophysiological effects produced by Tityus serrulatus toxin 1 on NaV-type isoforms., Peigneur S., Neuropharmacology. August 1, 2015; 95 269-77.
	Α- and β-subunit composition of voltage-gated sodium channels investigated with μ-conotoxins and the recently discovered μO§-conotoxin GVIIJ., Wilson MJ., J Neurophysiol. April 1, 2015; 113 (7): 2289-301.
	A novel µ-conopeptide, CnIIIC, exerts potent and preferential inhibition of NaV1.2/1.4 channels and blocks neuronal nicotinic acetylcholine receptors., Favreau P., Br J Pharmacol. July 1, 2012; 166 (5): 1654-68.
	Differential state-dependent modification of inactivation-deficient Nav1.6 sodium channels by the pyrethroid insecticides S-bioallethrin, tefluthrin and deltamethrin., McCavera SJ., Neurotoxicology. June 1, 2012; 33 (3): 384-90.
	Navβ subunits modulate the inhibition of Nav1.8 by the analgesic gating modifier μO-conotoxin MrVIB., Wilson MJ., J Pharmacol Exp Ther. August 1, 2011; 338 (2): 687-93.
	μ-Conotoxins that differentially block sodium channels NaV1.1 through 1.8 identify those responsible for action potentials in sciatic nerve., Wilson MJ., Proc Natl Acad Sci U S A. June 21, 2011; 108 (25): 10302-7.
	Phyla- and Subtype-Selectivity of CgNa, a Na Channel Toxin from the Venom of the Giant Caribbean Sea Anemone Condylactis Gigantea., Billen B., Front Pharmacol. November 23, 2010; 1 133.
	Molecular mechanism of allosteric modification of voltage-dependent sodium channels by local anesthetics., Arcisio-Miranda M., J Gen Physiol. November 1, 2010; 136 (5): 541-54.
	Induction of vertebrate regeneration by a transient sodium current., Tseng AS., J Neurosci. September 29, 2010; 30 (39): 13192-200.
	Optogenetic localization and genetic perturbation of saccade-generating neurons in zebrafish., Schoonheim PJ., J Neurosci. May 19, 2010; 30 (20): 7111-20.
	Electric currents in Xenopus tadpole tail regeneration., Reid B., Dev Biol. November 1, 2009; 335 (1): 198-207.
	The external pore loop interacts with S6 and S3-S4 linker in domain 4 to assume an essential role in gating control and anticonvulsant action in the Na(+) channel., Yang YC., J Gen Physiol. August 1, 2009; 134 (2): 95-113.
	Alpha-scorpion toxin impairs a conformational change that leads to fast inactivation of muscle sodium channels., Campos FV., J Gen Physiol. August 1, 2008; 132 (2): 251-63.
	A cation-pi interaction discriminates among sodium channels that are either sensitive or resistant to tetrodotoxin block., Santarelli VP., J Biol Chem. March 16, 2007; 282 (11): 8044-51.
	Structural basis for the voltage-gated Na+ channel selectivity of the scorpion alpha-like toxin BmK M1., Ye X., J Mol Biol. November 4, 2005; 353 (4): 788-803.
	State-dependent block of rat Nav1.4 sodium channels expressed in xenopus oocytes by pyrazoline-type insecticides., Silver K., Neurotoxicology. June 1, 2005; 26 (3): 397-406.
	A new type of scorpion Na+-channel-toxin-like polypeptide active on K+ channels., Srairi-Abid N., Biochem J. June 1, 2005; 388 (Pt 2): 455-64.
	A role for the carbohydrate portion of ginsenoside Rg3 in Na+ channel inhibition., Kim JH., Mol Cells. February 28, 2005; 19 (1): 137-42.
	The poison Dart frog's batrachotoxin modulates Nav1.8., Bosmans F., FEBS Lett. November 5, 2004; 577 (1-2): 245-8.
	State-dependent trapping of flecainide in the cardiac sodium channel., Ramos E., J Physiol. October 1, 2004; 560 (Pt 1): 37-49.
	Role of auxiliary beta1-, beta2-, and beta3-subunits and their interaction with Na(v)1.8 voltage-gated sodium channel., Vijayaragavan K., Biochem Biophys Res Commun. June 25, 2004; 319 (2): 531-40.
	Differential modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by the local anesthetic lidocaine., Chevrier P., Br J Pharmacol. June 1, 2004; 142 (3): 576-84.
	Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C., Vijayaragavan K., J Neurophysiol. April 1, 2004; 91 (4): 1556-69.
	Importance of the conserved aromatic residues in the scorpion alpha-like toxin BmK M1: the hydrophobic surface region revisited., Sun YM., J Biol Chem. June 27, 2003; 278 (26): 24125-31.
	Lidocaine block of neonatal Nav1.3 is differentially modulated by co-expression of beta1 and beta3 subunits., Lenkowski PW., Eur J Pharmacol. April 25, 2003; 467 (1-3): 23-30.
	Sodium channel heterologous expression in mammalian cells and the role of the endogenous beta1-subunits., Moran O., Neurosci Lett. January 23, 2003; 336 (3): 175-9.
	Point mutations in homology domain II modify the sensitivity of rat Nav1.8 sodium channels to the pyrethroid insecticide cismethrin., Soderlun DM., Neurotoxicology. December 1, 2001; 22 (6): 755-65.
	Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels., Vijayaragavan K., J Neurosci. October 15, 2001; 21 (20): 7909-18.

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