Papers associated with egg (and mre11)

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	Resection of DNA double-strand breaks activates Mre11-Rad50-Nbs1- and Rad9-Hus1-Rad1-dependent mechanisms that redundantly promote ATR checkpoint activation and end processing in Xenopus egg extracts., Tatsukawa K., Nucleic Acids Res. February 13, 2024;
	Profiling ubiquitin signalling with UBIMAX reveals DNA damage- and SCFβ-Trcp1-dependent ubiquitylation of the actin-organizing protein Dbn1., Colding-Christensen CS., Nat Commun. December 14, 2023; 14 (1): 8293.
	POLθ prevents MRE11-NBS1-CtIP-dependent fork breakage in the absence of BRCA2/RAD51 by filling lagging-strand gaps., Mann A., Mol Cell. November 17, 2022; 82 (22): 4218-4231.e8.
	MRN-dependent and independent pathways for recruitment of TOPBP1 to DNA double-strand breaks., Montales K., PLoS One. August 2, 2022; 17 (8): e0271905.
	Mre11 exonuclease activity promotes irreversible mitotic progression under replication stress., Hashimoto Y., Life Sci Alliance. March 15, 2022; 5 (6):
	Structure-function analysis of TOPBP1's role in ATR signaling using the DSB-mediated ATR activation in Xenopus egg extracts (DMAX) system., Montales K., Sci Rep. January 11, 2021; 11 (1): 467.
	RPA-coated single-stranded DNA promotes the ETAA1-dependent activation of ATR., Lyu K., Cell Cycle. April 1, 2019; 18 (8): 898-913.
	Fanconi-Anemia-Associated Mutations Destabilize RAD51 Filaments and Impair Replication Fork Protection., Zadorozhny K., Cell Rep. October 10, 2017; 21 (2): 333-340.
	Smarcal1-Mediated Fork Reversal Triggers Mre11-Dependent Degradation of Nascent DNA in the Absence of Brca2 and Stable Rad51 Nucleofilaments., Kolinjivadi AM., Mol Cell. September 7, 2017; 67 (5): 867-881.e7.
	Cip29 is phosphorylated following activation of the DNA damage response in Xenopus egg extracts., Holden J., PLoS One. July 10, 2017; 12 (7): e0181131.
	Mdc1 modulates the interaction between TopBP1 and the MRN complex during DNA damage checkpoint responses., Choi SH., Biochem Biophys Res Commun. October 7, 2016; 479 (1): 5-11.
	Xenopus Mcm10 is a CDK-substrate required for replication fork stability., Chadha GS., Cell Cycle. August 17, 2016; 15 (16): 2183-2195.
	The structure of ends determines the pathway choice and Mre11 nuclease dependency of DNA double-strand break repair., Liao S., Nucleic Acids Res. July 8, 2016; 44 (12): 5689-701.
	Suppression of DNA-damage checkpoint signaling by Rsk-mediated phosphorylation of Mre11., Chen C., Proc Natl Acad Sci U S A. December 17, 2013; 110 (51): 20605-10.
	The Mre11-Rad50-Nbs1 (MRN) complex has a specific role in the activation of Chk1 in response to stalled replication forks., Lee J., Mol Biol Cell. May 1, 2013; 24 (9): 1343-53.
	A role for the MRN complex in ATR activation via TOPBP1 recruitment., Duursma AM., Mol Cell. April 11, 2013; 50 (1): 116-22.
	The MRN-CtIP pathway is required for metaphase chromosome alignment., Rozier L., Mol Cell. March 28, 2013; 49 (6): 1097-107.
	Analysis of MRE11's function in the 5'-->3' processing of DNA double-strand breaks., Liao S., Nucleic Acids Res. May 1, 2012; 40 (10): 4496-506.
	Time-dependent predominance of nonhomologous DNA end-joining pathways during embryonic development in mice., Chiruvella KK., J Mol Biol. March 30, 2012; 417 (3): 197-211.
	Role for Rif1 in the checkpoint response to damaged DNA in Xenopus egg extracts., Kumar S., Cell Cycle. March 15, 2012; 11 (6): 1183-94.
	RAD51- and MRE11-dependent reassembly of uncoupled CMG helicase complex at collapsed replication forks., Hashimoto Y., Nat Struct Mol Biol. December 4, 2011; 19 (1): 17-24.
	Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair., Peterson SE., J Cell Biol. September 5, 2011; 194 (5): 705-20.
	CtIP interacts with TopBP1 and Nbs1 in the response to double-stranded DNA breaks (DSBs) in Xenopus egg extracts., Ramírez-Lugo JS., Cell Cycle. February 1, 2011; 10 (3): 469-80.
	Replication protein A promotes 5'-->3' end processing during homology-dependent DNA double-strand break repair., Yan H., J Cell Biol. January 24, 2011; 192 (2): 251-61.
	Rad51 protects nascent DNA from Mre11-dependent degradation and promotes continuous DNA synthesis., Hashimoto Y., Nat Struct Mol Biol. November 1, 2010; 17 (11): 1305-11.
	Xenopus DNA2 is a helicase/nuclease that is found in complexes with replication proteins And-1/Ctf4 and Mcm10 and DSB response proteins Nbs1 and ATM., Wawrousek KE., Cell Cycle. March 15, 2010; 9 (6): 1156-66.
	The Mre11/Rad50/Nbs1 complex functions in resection-based DNA end joining in Xenopus laevis., Taylor EM., Nucleic Acids Res. January 1, 2010; 38 (2): 441-54.
	CtIP links DNA double-strand break sensing to resection., You Z., Mol Cell. December 25, 2009; 36 (6): 954-69.
	The Mre11-Rad50-Nbs1 complex mediates activation of TopBP1 by ATM., Yoo HY., Mol Biol Cell. May 1, 2009; 20 (9): 2351-60.
	Ku80 removal from DNA through double strand break-induced ubiquitylation., Postow L., J Cell Biol. August 11, 2008; 182 (3): 467-79.
	Mre11-Rad50-Nbs1-dependent processing of DNA breaks generates oligonucleotides that stimulate ATM activity., Jazayeri A., EMBO J. July 23, 2008; 27 (14): 1953-62.
	ATM and ATR promote Mre11 dependent restart of collapsed replication forks and prevent accumulation of DNA breaks., Trenz K., EMBO J. April 19, 2006; 25 (8): 1764-74.
	Repair of double-strand breaks by nonhomologous end joining in the absence of Mre11., Di Virgilio M., J Cell Biol. December 5, 2005; 171 (5): 765-71.
	ATM activation and its recruitment to damaged DNA require binding to the C terminus of Nbs1., You Z., Mol Cell Biol. July 1, 2005; 25 (13): 5363-79.
	Mre11 assembles linear DNA fragments into DNA damage signaling complexes., Costanzo V., PLoS Biol. May 1, 2004; 2 (5): E110.

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