Papers associated with cav2

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	Dominant-negative effects of human P/Q-type Ca2+ channel mutations associated with episodic ataxia type 2., Jeng CJ, Chen YT, Chen YW, Tang CY., Am J Physiol Cell Physiol. April 1, 2006; 290 (4): C1209-20.
	Alternative splicing of the voltage-gated Ca2+ channel beta4 subunit creates a uniquely folded N-terminal protein binding domain with cell-specific expression in the cerebellar cortex., Vendel AC, Terry MD, Striegel AR, Iverson NM, Leuranguer V, Rithner CD, Lyons BA, Pickard GE, Tobet SA, Horne WA., J Neurosci. March 8, 2006; 26 (10): 2635-44.
	Localization of the activation gate of a voltage-gated Ca2+ channel., Xie C, Zhen XG, Yang J., J Gen Physiol. September 1, 2005; 126 (3): 205-12.
	Functional architecture of the inner pore of a voltage-gated Ca2+ channel., Zhen XG, Xie C, Fitzmaurice A, Schoonover CE, Orenstein ET, Yang J., J Gen Physiol. September 1, 2005; 126 (3): 193-204.
	Co-localization and interaction of organic anion transporter 1 with caveolin-2 in rat kidney., Kwak JO, Kim HW, Oh KJ, Kim DS, Han KO, Cha SH., Exp Mol Med. June 30, 2005; 37 (3): 204-12.
	Familial hemiplegic migraine type 1 mutations K1336E, W1684R, and V1696I alter Cav2.1 Ca2+ channel gating: evidence for beta-subunit isoform-specific effects., Müllner C, Broos LA, van den Maagdenberg AM, Striessnig J., J Biol Chem. December 10, 2004; 279 (50): 51844-50.
	A Purkinje cell specific GoLoco domain protein, L7/Pcp-2, modulates receptor-mediated inhibition of Cav2.1 Ca2+ channels in a dose-dependent manner., Kinoshita-Kawada M, Oberdick J, Xi Zhu M., Brain Res Mol Brain Res. December 6, 2004; 132 (1): 73-86.
	Dysfunction of the brain calcium channel CaV2.1 in absence epilepsy and episodic ataxia., Imbrici P, Jaffe SL, Eunson LH, Davies NP, Herd C, Robertson R, Kullmann DM, Hanna MG., Brain. December 1, 2004; 127 (Pt 12): 2682-92.
	Negatively charged residues in the N-terminal of the AID helix confer slow voltage dependent inactivation gating to CaV1.2., Dafi O, Berrou L, Dodier Y, Raybaud A, Sauvé R, Parent L., Biophys J. November 1, 2004; 87 (5): 3181-92.
	Ca2+-dependent interaction of BAPTA with phospholipids., Rousset M, Cens T, Vanmau N, Charnet P., FEBS Lett. October 8, 2004; 576 (1-2): 41-5.
	The alpha2delta auxiliary subunit reduces affinity of omega-conotoxins for recombinant N-type (Cav2.2) calcium channels., Mould J, Yasuda T, Schroeder CI, Beedle AM, Doering CJ, Zamponi GW, Adams DJ, Lewis RJ., J Biol Chem. August 13, 2004; 279 (33): 34705-14.
	Overexpressed Ca(v)beta3 inhibits N-type (Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation., Yasuda T, Lewis RJ, Adams DJ., J Gen Physiol. April 1, 2004; 123 (4): 401-16.
	The caveolin proteins., Williams TM, Lisanti MP., Genome Biol. January 1, 2004; 5 (3): 214.
	CaVbeta subunit-mediated up-regulation of CaV2.2 currents triggered by D2 dopamine receptor activation., Cantí C, Dolphin AC., Neuropharmacology. November 1, 2003; 45 (6): 814-27.
	Specific sites in the Beta Interaction Domain of a schistosome Ca2+ channel beta subunit are key to its role in sensitivity to the anti-schistosomal drug praziquantel., Kohn AB, Roberts-Misterly JM, Anderson PA, Khan N, Greenberg RM., Parasitology. October 1, 2003; 127 (Pt 4): 349-56.
	Human neuronal stargazin-like proteins, gamma2, gamma3 and gamma4; an investigation of their specific localization in human brain and their influence on CaV2.1 voltage-dependent calcium channels expressed in Xenopus oocytes., Moss FJ, Dolphin AC, Clare JJ., BMC Neurosci. September 23, 2003; 4 23.
	The C2A domain of synaptotagmin alters the kinetics of voltage-gated Ca2+ channels Ca(v)1.2 (Lc-type) and Ca(v)2.3 (R-type)., Cohen R, Elferink LA, Atlas D., J Biol Chem. March 14, 2003; 278 (11): 9258-66.
	Alternative splicing of a beta4 subunit proline-rich motif regulates voltage-dependent gating and toxin block of Cav2.1 Ca2+ channels., Helton TD, Kojetin DJ, Cavanagh J, Horne WA., J Neurosci. November 1, 2002; 22 (21): 9331-9.
	The interaction between the I-II loop and the III-IV loop of Cav2.1 contributes to voltage-dependent inactivation in a beta -dependent manner., Geib S, Sandoz G, Cornet V, Mabrouk K, Fund-Saunier O, Bichet D, Villaz M, Hoshi T, Sabatier JM, De Waard M., J Biol Chem. March 22, 2002; 277 (12): 10003-13.
	Functional consequences of P/Q-type Ca2+ channel Cav2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia., Wappl E, Koschak A, Poteser M, Sinnegger MJ, Walter D, Eberhart A, Groschner K, Glossmann H, Kraus RL, Grabner M, Striessnig J., J Biol Chem. March 1, 2002; 277 (9): 6960-6.
	Functional expression and characterization of a voltage-gated CaV1.3 (alpha1D) calcium channel subunit from an insulin-secreting cell line., Scholze A, Plant TD, Dolphin AC, Nürnberg B., Mol Endocrinol. July 1, 2001; 15 (7): 1211-21.
	Molecular determinants of inactivation within the I-II linker of alpha1E (CaV2.3) calcium channels., Berrou L, Bernatchez G, Parent L., Biophys J. January 1, 2001; 80 (1): 215-28.

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