Papers associated with mink1

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	Nucleotide sequence of cDNA coding for mink proopiomelanocortin (POMC) and its comparative analysis with POMC mRNA primary structures from pituitaries of other animal species and man., Mertvetsov NP, Karginov VA, Golovin SJa, Bondar AA, Morozov IV, Zelenin SM, Blinov VM., Biomed Biochim Acta. January 1, 1991; 50 (9): 1057-64.
	Modulation by cAMP of a slowly activating potassium channel expressed in Xenopus oocytes., Blumenthal EM, Kaczmarek LK., J Neurosci. January 1, 1992; 12 (1): 290-6.
	Structure and regulation of the MinK potassium channel., Blumenthal EM, Kaczmarek LK., Neurochem Res. September 1, 1992; 17 (9): 869-76.
	Inward rectification of the minK potassium channel., Blumenthal EM, Kaczmarek LK., J Membr Biol. October 1, 1993; 136 (1): 23-9.
	Expression of a minimal K+ channel protein in mammalian cells and immunolocalization in guinea pig heart., Freeman LC, Kass RS., Circ Res. November 1, 1993; 73 (5): 968-73.
	Effects of [Ca2+]i and temperature on minK channels expressed in Xenopus oocytes., Busch AE, Lang F., FEBS Lett. November 15, 1993; 334 (2): 221-4.
	Time dependent changes in biophysical properties of minK channels expressed in Xenopus oocytes., Busch AE, Lang F., Biochem Biophys Res Commun. December 15, 1993; 197 (2): 473-7.
	The minK potassium channel exists in functional and nonfunctional forms when expressed in the plasma membrane of Xenopus oocytes., Blumenthal EM, Kaczmarek LK., J Neurosci. May 1, 1994; 14 (5 Pt 2): 3097-105.
	Secondary structure and membrane localization of synthetic segments and a truncated form of the IsK (minK) protein., Ben-Efraim I, Strahilevitz J, Bach D, Shai Y., Biochemistry. June 7, 1994; 33 (22): 6966-73.
	Subunit composition of minK potassium channels., Wang KW, Goldstein SA., Neuron. June 1, 1995; 14 (6): 1303-9.
	Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel., Sanguinetti MC, Curran ME, Zou A, Shen J, Spector PS, Atkinson DL, Keating MT., Nature. November 7, 1996; 384 (6604): 80-3.
	K(V)LQT1 and lsK (minK) proteins associate to form the I(Ks) cardiac potassium current., Barhanin J, Lesage F, Guillemare E, Fink M, Lazdunski M, Romey G., Nature. November 7, 1996; 384 (6604): 78-80.
	IK of rabbit ventricle is composed of two currents: evidence for IKs., Salata JJ, Jurkiewicz NK, Jow B, Folander K, Guinosso PJ, Raynor B, Swanson R, Fermini B., Am J Physiol. December 1, 1996; 271 (6 Pt 2): H2477-89.
	Inhibition of minK protein induced K+ channels in Xenopus oocytes by estrogens., Waldegger S, Lang U, Herzer T, Suessbrich H, Binder K, Lepple-Wienhues A, Nagl U, Paulmichl M, Franz HB, Kiesl L, Lang F, Busch AE., Naunyn Schmiedebergs Arch Pharmacol. December 1, 1996; 354 (6): 698-702.
	Role of the ISK protein in the IminK channel complex., Busch AE, Suessbrich H., Trends Pharmacol Sci. January 1, 1997; 18 (1): 26-9.
	MinK potassium channels are heteromultimeric complexes., Tai KK, Wang KW, Goldstein SA., J Biol Chem. January 17, 1997; 272 (3): 1654-8.
	Properties and regulation of the minK potassium channel protein., Kaczmarek LK, Blumenthal EM., Physiol Rev. July 1, 1997; 77 (3): 627-41.
	A minK-HERG complex regulates the cardiac potassium current I(Kr)., McDonald TV, Yu Z, Ming Z, Palma E, Meyers MB, Wang KW, Goldstein SA, Fishman GI., Nature. July 17, 1997; 388 (6639): 289-92.
	Dominant-negative KvLQT1 mutations underlie the LQT1 form of long QT syndrome., Shalaby FY, Levesque PC, Yang WP, Little WA, Conder ML, Jenkins-West T, Blanar MA., Circulation. September 16, 1997; 96 (6): 1733-6.
	Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE, Eigenberger B, Jurkiewicz NK, Salata JJ, Pica A, Suessbrich H, Lang F., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.
	The conduction pore of a cardiac potassium channel., Tai KK, Goldstein SA., Nature. February 5, 1998; 391 (6667): 605-8.
	Effects of the chromanol 293B, a selective blocker of the slow, component of the delayed rectifier K+ current, on repolarization in human and guinea pig ventricular myocytes., Bosch RF, Gaspo R, Busch AE, Lang HJ, Li GR, Nattel S., Cardiovasc Res. May 1, 1998; 38 (2): 441-50.
	Gating of I(sK) channels expressed in Xenopus oocytes., Tzounopoulos T, Maylie J, Adelman JP., Biophys J. May 1, 1998; 74 (5): 2299-305.
	A novel benzodiazepine that activates cardiac slow delayed rectifier K+ currents., Salata JJ, Jurkiewicz NK, Wang J, Evans BE, Orme HT, Sanguinetti MC., Mol Pharmacol. July 1, 1998; 54 (1): 220-30.
	Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits., Tristani-Firouzi M, Sanguinetti MC., J Physiol. July 1, 1998; 510 ( Pt 1) 37-45.
	Functional expression of two KvLQT1-related potassium channels responsible for an inherited idiopathic epilepsy., Yang WP, Levesque PC, Little WA, Conder ML, Ramakrishnan P, Neubauer MG, Blanar MA., J Biol Chem. July 31, 1998; 273 (31): 19419-23.
	Single-channel properties of IKs potassium channels., Yang Y, Sigworth FJ., J Gen Physiol. December 1, 1998; 112 (6): 665-78.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F, Goldstein SA., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
	Increase of the single-channel conductance of KvLQT1 potassium channels induced by the association with minK., Pusch M., Pflugers Arch. December 1, 1998; 437 (1): 172-4.
	Receptors for polytropic and xenotropic mouse leukaemia viruses encoded by a single gene at Rmc1., Yang YL, Guo L, Xu S, Holland CA, Kitamura T, Hunter K, Cunningham JM., Nat Genet. February 1, 1999; 21 (2): 216-9.
	PKC modulation of minK current involves multiple phosphorylation sites., Lo CF, Numann R., Ann N Y Acad Sci. April 30, 1999; 868 431-3.
	Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with minK subunits., Franqueza L, Lin M, Shen J, Splawski I, Keating MT, Sanguinetti MC., J Biol Chem. July 23, 1999; 274 (30): 21063-70.
	Cellular dysfunction of LQT5-minK mutants: abnormalities of IKs, IKr and trafficking in long QT syndrome., Bianchi L, Shen Z, Dennis AT, Priori SG, Napolitano C, Ronchetti E, Bryskin R, Schwartz PJ, Brown AM., Hum Mol Genet. August 1, 1999; 8 (8): 1499-507.
	A constitutively open potassium channel formed by KCNQ1 and KCNE3., Schroeder BC, Waldegger S, Fehr S, Bleich M, Warth R, Greger R, Jentsch TJ., Nature. January 13, 2000; 403 (6766): 196-9.
	Molecular basis for differential sensitivity of KCNQ and I(Ks) channels to the cognitive enhancer XE991., Wang HS, Brown BS, McKinnon D, Cohen IS., Mol Pharmacol. June 1, 2000; 57 (6): 1218-23.
	MinK endows the I(Ks) potassium channel pore with sensitivity to internal tetraethylammonium., Sesti F, Tai KK, Goldstein SA., Biophys J. September 1, 2000; 79 (3): 1369-78.
	MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR, George AL., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
	Functional coupling of human beta 3-adrenoreceptors to the KvLQT1/MinK potassium channel., Kathöfer S, Zhang W, Karle C, Thomas D, Schoels W, Kiehn J., J Biol Chem. September 1, 2000; 275 (35): 26743-7.
	Molecular impact of MinK on the enantiospecific block of I(Ks) by chromanols., Lerche C, Seebohm G, Wagner CI, Scherer CR, Dehmelt L, Abitbol I, Gerlach U, Brendel J, Attali B, Busch AE., Br J Pharmacol. December 1, 2000; 131 (8): 1503-6.
	KCNE2 confers background current characteristics to the cardiac KCNQ1 potassium channel., Tinel N, Diochot S, Borsotto M, Lazdunski M, Barhanin J., EMBO J. December 1, 2000; 19 (23): 6326-30.
	Identification of specific pore residues mediating KCNQ1 inactivation. A novel mechanism for long QT syndrome., Seebohm G, Scherer CR, Busch AE, Lerche C., J Biol Chem. April 27, 2001; 276 (17): 13600-5.
	minK-related peptide 1 associates with Kv4.2 and modulates its gating function: potential role as beta subunit of cardiac transient outward channel?, Zhang M, Jiang M, Tseng GN., Circ Res. May 25, 2001; 88 (10): 1012-9.
	MinK-related peptide 1: A beta subunit for the HCN ion channel subunit family enhances expression and speeds activation., Yu H, Wu J, Potapova I, Wymore RT, Holmes B, Zuckerman J, Pan Z, Wang H, Shi W, Robinson RB, El-Maghrabi MR, Benjamin W, Dixon J, McKinnon D, Cohen IS, Wymore R., Circ Res. June 22, 2001; 88 (12): E84-7.
	Location and orientation of minK within the I(Ks) potassium channel complex., Tapper AR, George AL., J Biol Chem. October 12, 2001; 276 (41): 38249-54.
	Inhibition of cardiac potassium currents by pentobarbital., Bachmann A, Mueller S, Kopp K, Brueggemann A, Suessbrich H, Gerlach U, Busch AE., Naunyn Schmiedebergs Arch Pharmacol. January 1, 2002; 365 (1): 29-37.
	A truncated splice variant of KCNQ1 cloned from rat heart., Yamada Y, Chen X, Kobayashi T, Kamada Y, Nagashima M, Tsutsuura M, Seki S, Yamakage M, Namiki A, Tohse N., Biochem Biophys Res Commun. June 7, 2002; 294 (2): 199-204.
	Inhibitory effects of volatile anesthetics on currents produced on heterologous expression of KvLQT1 and minK in Xenopus oocytes., Chen X, Yamakage M, Yamada Y, Tohse N, Namiki A., Vascul Pharmacol. July 1, 2002; 39 (1-2): 33-8.
	Clinical and electrophysiological characterization of a novel mutation (F193L) in the KCNQ1 gene associated with long QT syndrome., Yamaguchi M, Shimizu M, Ino H, Terai H, Hayashi K, Mabuchi H, Hoshi N, Higashida H., Clin Sci (Lond). April 1, 2003; 104 (4): 377-82.
	RNA interference reveals that endogenous Xenopus MinK-related peptides govern mammalian K+ channel function in oocyte expression studies., Anantharam A, Lewis A, Panaghie G, Gordon E, McCrossan ZA, Lerner DJ, Abbott GW., J Biol Chem. April 4, 2003; 278 (14): 11739-45.
	Pore- and state-dependent cadmium block of I(Ks) channels formed with MinK-55C and wild-type KCNQ1 subunits., Chen H, Sesti F, Goldstein SA., Biophys J. June 1, 2003; 84 (6): 3679-89.

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