Mouse (77 sources):
abnormal T-helper 1 physiology,
abnormal angiogenesis,
abnormal associative learning,
abnormal erythropoiesis,
abnormal fetal cardiomyocyte morphology,
abnormal fetal cardiomyocyte proliferation,
abnormal long bone epiphyseal plate proliferative zone,
abnormal lung development,
abnormal lung epithelium morphology,
abnormal muscle fiber morphology,
abnormal myoblast differentiation,
abnormal myoblast fusion,
abnormal placenta development,
abnormal placenta labyrinth morphology,
abnormal placenta morphology,
abnormal placenta vasculature,
abnormal placental labyrinth vasculature morphology,
abnormal pulmonary alveolus epithelial cell morphology,
abnormal response to cardiac infarction,
abnormal trophoblast layer morphology,
abnormal tumor incidence,
abnormal vitelline vascular remodeling,
absent myocardial trabeculae,
absent placental labyrinth,
behavior/neurological phenotype,
cardiac fibrosis,
cardiac hypertrophy,
cardiac interstitial fibrosis,
cardiovascular system phenotype,
cellular phenotype,
decreased T cell proliferation,
decreased body length,
decreased body size,
decreased cardiac muscle contractility,
decreased circulating glucose level,
decreased interferon-alpha secretion,
decreased interferon-gamma secretion,
decreased long bone epiphyseal plate size,
decreased myocardial infarct size,
decreased pancreatic beta cell proliferation,
decreased physiological sensitivity to xenobiotic,
decreased placental labyrinth size,
decreased spongiotrophoblast size,
decreased trophoblast giant cell number,
decreased ventricle muscle contractility,
decreased width of hypertrophic chondrocyte zone,
embryonic growth retardation,
embryonic lethality during organogenesis,
embryonic lethality during organogenesis, complete penetrance,
embryonic lethality during organogenesis, incomplete penetrance,
enlarged lung,
enlarged myocardial fiber,
hematopoietic system phenotype,
homeostasis/metabolism phenotype,
impaired placental function,
increased cardiomyocyte apoptosis,
increased cell proliferation,
increased double-negative T cell number,
increased embryonic tissue cell apoptosis,
increased heart weight,
increased lung adenoma incidence,
increased lung tumor incidence,
increased myoblast proliferation,
increased pancreatic beta cell proliferation,
increased response of heart to induced stress,
increased sensitivity to induced morbidity/mortality,
lethality throughout fetal growth and development, complete penetrance,
muscle phenotype,
no abnormal phenotype detected,
pale liver,
pale yolk sac,
poor circulation,
postnatal lethality, incomplete penetrance,
premature death,
prenatal lethality, complete penetrance,
small liver,
thin myocardium
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