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Akizuki,
Impaired solute accumulation in inner medulla of Clcnk1-/- mice kidney.
2001, Pubmed
Akizuki,
Impaired solute accumulation in inner medulla of Clcnk1-/- mice kidney.
2001,
Pubmed
Barlassina,
Common genetic variants and haplotypes in renal CLCNKA gene are associated to salt-sensitive hypertension.
2007,
Pubmed
Belachgar,
Triflocin, a novel inhibitor for the Na-HCO3 symport in the proximal tubule.
1994,
Pubmed
Birkenhäger,
Mutation of BSND causes Bartter syndrome with sensorineural deafness and kidney failure.
2001,
Pubmed
Dhanaraj,
Structural studies of analgesics and their interactions. XII. Structure and interactions of anti-inflammatory fenamates. A concerted crystallographic and theoretical conformational study.
1988,
Pubmed
Estévez,
Barttin is a Cl- channel beta-subunit crucial for renal Cl- reabsorption and inner ear K+ secretion.
2001,
Pubmed
,
Xenbase
Fong,
CLC-K channels: if the drug fits, use it.
2004,
Pubmed
Jeck,
Activating mutation of the renal epithelial chloride channel ClC-Kb predisposing to hypertension.
2004,
Pubmed
,
Xenbase
Jentsch,
Chloride transport in the kidney: lessons from human disease and knockout mice.
2005,
Pubmed
Kokubo,
Association analysis between hypertension and CYBA, CLCNKB, and KCNMB1 functional polymorphisms in the Japanese population--the Suita Study.
2005,
Pubmed
Konrad,
Mutations in the chloride channel gene CLCNKB as a cause of classic Bartter syndrome.
2000,
Pubmed
Liantonio,
Molecular requisites for drug binding to muscle CLC-1 and renal CLC-K channel revealed by the use of phenoxy-alkyl derivatives of 2-(p-chlorophenoxy)propionic acid.
2002,
Pubmed
,
Xenbase
Liantonio,
Investigations of pharmacologic properties of the renal CLC-K1 chloride channel co-expressed with barttin by the use of 2-(p-Chlorophenoxy)propionic acid derivatives and other structurally unrelated chloride channels blockers.
2004,
Pubmed
,
Xenbase
Liantonio,
Activation and inhibition of kidney CLC-K chloride channels by fenamates.
2006,
Pubmed
,
Xenbase
Markovic,
The structure of the cytoplasmic domain of the chloride channel ClC-Ka reveals a conserved interaction interface.
2007,
Pubmed
Picollo,
Mechanism of interaction of niflumic acid with heterologously expressed kidney CLC-K chloride channels.
2007,
Pubmed
,
Xenbase
Picollo,
Molecular determinants of differential pore blocking of kidney CLC-K chloride channels.
2004,
Pubmed
Picollo,
Chloride/proton antiporter activity of mammalian CLC proteins ClC-4 and ClC-5.
2005,
Pubmed
Pusch,
Pharmacological characterization of chloride channels belonging to the ClC family by the use of chiral clofibric acid derivatives.
2000,
Pubmed
,
Xenbase
Scheel,
Voltage-dependent electrogenic chloride/proton exchange by endosomal CLC proteins.
2005,
Pubmed
,
Xenbase
Scholl,
Barttin modulates trafficking and function of ClC-K channels.
2006,
Pubmed
Sile,
Molecular physiology of renal ClC chloride channels/transporters.
2006,
Pubmed
Simon,
Mutations in the chloride channel gene, CLCNKB, cause Bartter's syndrome type III.
1997,
Pubmed
Thiemann,
A chloride channel widely expressed in epithelial and non-epithelial cells.
1992,
Pubmed
,
Xenbase
Uchida,
Function of chloride channels in the kidney.
2005,
Pubmed
Waldegger,
Barttin increases surface expression and changes current properties of ClC-K channels.
2002,
Pubmed
,
Xenbase
