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Novel truncating mutations in CTNND1 cause a dominant craniofacial and cardiac syndrome. , Alharatani R., Hum Mol Genet. July 21, 2020; 29 (11): 1900-1921.
Structural determinants at the M2 muscarinic receptor modulate the RGS4- GIRK response to pilocarpine by impairment of the receptor voltage sensitivity. , Chen IS., Sci Rep. July 21, 2017; 7 (1): 6110.
Neural crest specification by inhibition of the ROCK/Myosin II pathway. , Kim K., Stem Cells. March 1, 2015; 33 (3): 674-85.
Optogenetic manipulation of cGMP in cells and animals by the tightly light-regulated guanylyl-cyclase opsin CyclOp. , Gao S., Nat Commun. January 19, 2015; 6 8046.
The function of p120 catenin in filopodial growth and synaptic vesicle clustering in neurons. , Chen C ., Mol Biol Cell. July 1, 2012; 23 (14): 2680-91.
TASK1 (K(2P)3.1) K(+) channel inhibition by endothelin-1 is mediated through Rho kinase-dependent phosphorylation. , Seyler C., Br J Pharmacol. March 1, 2012; 165 (5): 1467-75.
SHP-2 acts via ROCK to regulate the cardiac actin cytoskeleton. , Langdon Y ., Development. March 1, 2012; 139 (5): 948-57.
The function of cortactin in the clustering of acetylcholine receptors at the vertebrate neuromuscular junction. , Madhavan R., PLoS One. December 29, 2009; 4 (12): e8478.
tBid mediated activation of the mitochondrial death pathway leads to genetic ablation of the lens in Xenopus laevis. , Du Pasquier D., Genesis. January 1, 2007; 45 (1): 1-10.
Protein phosphatase activity is necessary for myofibrillogenesis. , Terry M., Cell Biochem Biophys. January 1, 2006; 45 (3): 265-78.
Inhibition of Rho family GTPases by Rho GDP dissociation inhibitor disrupts cardiac morphogenesis and inhibits cardiomyocyte proliferation. , Wei L., Development. April 1, 2002; 129 (7): 1705-14.
Induction of avian cardiac myogenesis by anterior endoderm. , Schultheiss TM., Development. December 1, 1995; 121 (12): 4203-14.
Modulation of cardiac Na+ channel expression in Xenopus oocytes by beta 1 subunits. , Qu Y., J Biol Chem. October 27, 1995; 270 (43): 25696-701.
Specificity for block by saxitoxin and divalent cations at a residue which determines sensitivity of sodium channel subtypes to guanidinium toxins. , Favre I., J Gen Physiol. August 1, 1995; 106 (2): 203-29.