Papers associated with skeletal element (and trpv4)

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	Trpc1 as the Missing Link Between the Bmp and Ca2+ Signalling Pathways During Neural Specification in Amphibians., Néant I., Sci Rep. November 5, 2019; 9 (1): 16049.
	Human mutations highlight an intersubunit cation-π bond that stabilizes the closed but not open or inactivated states of TRPV channels., Teng J., Proc Natl Acad Sci U S A. May 7, 2019; 116 (19): 9410-9416.
	L596-W733 bond between the start of the S4-S5 linker and the TRP box stabilizes the closed state of TRPV4 channel., Teng J., Proc Natl Acad Sci U S A. March 17, 2015; 112 (11): 3386-91.
	Yeast luminometric and Xenopus oocyte electrophysiological examinations of the molecular mechanosensitivity of TRPV4., Teng J., J Vis Exp. December 31, 2013; (82):
	Increased basal activity is a key determinant in the severity of human skeletal dysplasia caused by TRPV4 mutations., Loukin S., PLoS One. May 3, 2011; 6 (5): e19533.
	Wild-type and brachyolmia-causing mutant TRPV4 channels respond directly to stretch force., Loukin S., J Biol Chem. August 27, 2010; 285 (35): 27176-81.
	Xenopus TRPN1 (NOMPC) localizes to microtubule-based cilia in epithelial cells, including inner-ear hair cells., Shin JB., Proc Natl Acad Sci U S A. August 30, 2005; 102 (35): 12572-7.

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