Papers associated with oocyte (and kcnd2)

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	Nocturnal Atrial Fibrillation Caused by Mutation in KCND2, Encoding Pore-Forming (α) Subunit of the Cardiac Kv4.2 Potassium Channel., Drabkin M., Circ Genom Precis Med. November 1, 2018; 11 (11): e002293.
	S-glutathionylation of an auxiliary subunit confers redox sensitivity to Kv4 channel inactivation., Jerng HH., PLoS One. January 1, 2014; 9 (3): e93315.
	Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1., Aidery P., Gene. December 10, 2012; 511 (1): 26-33.
	Modeling-independent elucidation of inactivation pathways in recombinant and native A-type Kv channels., Fineberg JD., J Gen Physiol. November 1, 2012; 140 (5): 513-27.
	Dynamic coupling of voltage sensor and gate involved in closed-state inactivation of kv4.2 channels., Barghaan J., J Gen Physiol. February 1, 2009; 133 (2): 205-24.
	A dipeptidyl aminopeptidase-like protein remodels gating charge dynamics in Kv4.2 channels., Dougherty K., J Gen Physiol. December 1, 2006; 128 (6): 745-53.
	Modulation of Kv4.2 channel expression and gating by dipeptidyl peptidase 10 (DPP10)., Jerng HH., Biophys J. October 1, 2004; 87 (4): 2380-96.
	minK-related peptide 1 associates with Kv4.2 and modulates its gating function: potential role as beta subunit of cardiac transient outward channel?, Zhang M., Circ Res. May 25, 2001; 88 (10): 1012-9.

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