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Summary Anatomy Item Literature (17693) Expression Attributions Wiki
XB-ANAT-504

Papers associated with tissue (and chrnb1)

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Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome., Gomez CM., J Neurosci. August 1, 2002; 22 (15): 6447-57.


Slow-channel transgenic mice: a model of postsynaptic organellar degeneration at the neuromuscular junction., Gomez CM., J Neurosci. June 1, 1997; 17 (11): 4170-9.


Mutations in different functional domains of the human muscle acetylcholine receptor alpha subunit in patients with the slow-channel congenital myasthenic syndrome., Croxen R., Hum Mol Genet. May 1, 1997; 6 (5): 767-74.


Former neuritic pathways containing endogenous neural agrin have high synaptogenic activity., Cohen MW., Dev Biol. February 1, 1995; 167 (2): 458-68.              

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