Papers associated with whole organism (and kcnj5)

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	Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation., Mesirca P., Nat Commun. August 21, 2014; 5 4664.
	Differential effects of genetically-encoded Gβγ scavengers on receptor-activated and basal Kir3.1/Kir3.4 channel current in rat atrial myocytes., Kienitz MC., Cell Signal. June 1, 2014; 26 (6): 1182-1192.
	A Kir3.4 mutation causes Andersen-Tawil syndrome by an inhibitory effect on Kir2.1., Kokunai Y., Neurology. March 25, 2014; 82 (12): 1058-64.
	RGS4 regulates partial agonism of the M2 muscarinic receptor-activated K+ currents., Chen IS., J Physiol. March 15, 2014; 592 (6): 1237-48.
	Characterization of a novel somatic KCNJ5 mutation delI157 in an aldosterone-producing adenoma., Murthy M., J Hypertens. September 1, 2012; 30 (9): 1827-33.
	Crystal structure of the mammalian GIRK2 K+ channel and gating regulation by G proteins, PIP2, and sodium., Whorton MR., Cell. September 30, 2011; 147 (1): 199-208.
	Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants., Kobayashi T., PLoS One. January 1, 2011; 6 (12): e28208.
	Characterizations of a loss-of-function mutation in the Kir3.4 channel subunit., Calloe K., Biochem Biophys Res Commun. December 28, 2007; 364 (4): 889-95.
	Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes., Kobayashi T., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.
	Mutation of critical GIRK subunit residues disrupts N- and C-termini association and channel function., Sarac R., J Neurosci. February 16, 2005; 25 (7): 1836-46.
	RFamide-related peptides signal through the neuropeptide FF receptor and regulate pain-related responses in the rat., Pertovaara A., Neuroscience. January 1, 2005; 134 (3): 1023-32.
	Molecular determinants responsible for differential cellular distribution of G protein-gated inwardly rectifying K+ channels., Mirshahi T., J Biol Chem. March 19, 2004; 279 (12): 11890-7.
	Mechanosensitivity of GIRK channels is mediated by protein kinase C-dependent channel-phosphatidylinositol 4,5-bisphosphate interaction., Zhang L., J Biol Chem. February 20, 2004; 279 (8): 7037-47.
	Early embryonic expression of ion channels and pumps in chick and Xenopus development., Rutenberg J., Dev Dyn. December 1, 2002; 225 (4): 469-84.
	G protein-gated inwardly rectifying potassium channels are targets for volatile anesthetics., Weigl LG., Mol Pharmacol. August 1, 2001; 60 (2): 282-9.
	Identification of G protein-coupled, inward rectifier potassium channel gene products from the rat anterior pituitary gland., Gregerson KA., Endocrinology. July 1, 2001; 142 (7): 2820-32.
	G-protein mediated gating of inward-rectifier K+ channels., Mark MD., Eur J Biochem. October 1, 2000; 267 (19): 5830-6.
	Heterologous facilitation of G protein-activated K(+) channels by beta-adrenergic stimulation via cAMP-dependent protein kinase., Müllner C., J Gen Physiol. May 1, 2000; 115 (5): 547-58.
	Probing the G-protein regulation of GIRK1 and GIRK4, the two subunits of the KACh channel, using functional homomeric mutants., Vivaudou M., J Biol Chem. December 12, 1997; 272 (50): 31553-60.
	Activation of heteromeric G protein-gated inward rectifier K+ channels overexpressed by adenovirus gene transfer inhibits the excitability of hippocampal neurons., Ehrengruber MU., Proc Natl Acad Sci U S A. June 24, 1997; 94 (13): 7070-5.
	Control of channel activity through a unique amino acid residue of a G protein-gated inwardly rectifying K+ channel subunit., Chan KW., Proc Natl Acad Sci U S A. November 26, 1996; 93 (24): 14193-8.
	Na+ activation of the muscarinic K+ channel by a G-protein-independent mechanism., Sui JL., J Gen Physiol. November 1, 1996; 108 (5): 381-91.
	Specificity of coupling of muscarinic receptor isoforms to a novel chick inward-rectifying acetylcholine-sensitive K+ channel., Gadbut AP., J Biol Chem. March 15, 1996; 271 (11): 6398-402.
	Localization and interaction of epitope-tagged GIRK1 and CIR inward rectifier K+ channel subunits., Kennedy ME., Neuropharmacology. January 1, 1996; 35 (7): 831-9.

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