Papers associated with whole organism (and kcnd2)

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	Modeling-independent elucidation of inactivation pathways in recombinant and native A-type Kv channels., Fineberg JD., J Gen Physiol. November 1, 2012; 140 (5): 513-27.
	Mutations in IRX5 impair craniofacial development and germ cell migration via SDF1., Bonnard C., Nat Genet. May 13, 2012; 44 (6): 709-13.
	The dipeptidyl-peptidase-like protein DPP6 determines the unitary conductance of neuronal Kv4.2 channels., Kaulin YA., J Neurosci. March 11, 2009; 29 (10): 3242-51.
	Kv4.2 is a locus for PKC and ERK/MAPK cross-talk., Schrader LA., Biochem J. February 1, 2009; 417 (3): 705-15.
	Interaction of syntaxin 1A with the N-terminus of Kv4.2 modulates channel surface expression and gating., Yamakawa T., Biochemistry. September 25, 2007; 46 (38): 10942-9.
	Molecular and functional characterization of Kv4.2 and KChIP2 expressed in the porcine left ventricle., Schultz JH., Pflugers Arch. May 1, 2007; 454 (2): 195-207.
	Diminished Kv4.2/3 but not KChIP2 levels reduce the cardiac transient outward K+ current in spontaneously hypertensive rats., Goltz D., Cardiovasc Res. April 1, 2007; 74 (1): 85-95.
	ERK/MAPK regulates the Kv4.2 potassium channel by direct phosphorylation of the pore-forming subunit., Schrader LA., Am J Physiol Cell Physiol. March 1, 2006; 290 (3): C852-61.
	Multiprotein assembly of Kv4.2, KChIP3 and DPP10 produces ternary channel complexes with ISA-like properties., Jerng HH., J Physiol. November 1, 2005; 568 (Pt 3): 767-88.
	Ito channels are octomeric complexes with four subunits of each Kv4.2 and K+ channel-interacting protein 2., Kim LA., J Biol Chem. February 13, 2004; 279 (7): 5549-54.
	PKA modulation of Kv4.2-encoded A-type potassium channels requires formation of a supramolecular complex., Schrader LA., J Neurosci. December 1, 2002; 22 (23): 10123-33.
	minK-related peptide 1 associates with Kv4.2 and modulates its gating function: potential role as beta subunit of cardiac transient outward channel?, Zhang M., Circ Res. May 25, 2001; 88 (10): 1012-9.
	Nicotine is a potent blocker of the cardiac A-type K(+) channels. Effects on cloned Kv4.3 channels and native transient outward current., Wang H., Circulation. September 5, 2000; 102 (10): 1165-71.
	[The expression of arrhythmic related genes on Xenopus oocytes for evaluation of class III antiarrhythmic drugs from ocean active material]., Xu DH., Yi Chuan Xue Bao. January 1, 2000; 27 (3): 195-201.
	Effect of Cd2+ on Kv4.2 and Kv1.4 expressed in Xenopus oocytes and on the transient outward currents in rat and rabbit ventricular myocytes., Wickenden AD., Cell Physiol Biochem. January 1, 1999; 9 (1): 11-28.
	Unexpected and differential effects of Cl- channel blockers on the Kv4.3 and Kv4.2 K+ channels. Implications for the study of the I(to2) current., Wang HS., Circ Res. November 1, 1997; 81 (5): 711-8.
	Specific block of cloned Herg channels by clofilium and its tertiary analog LY97241., Suessbrich H., FEBS Lett. September 8, 1997; 414 (2): 435-8.
	Heteropodatoxins: peptides isolated from spider venom that block Kv4.2 potassium channels., Sanguinetti MC., Mol Pharmacol. March 1, 1997; 51 (3): 491-8.
	Reverse use dependence of Kv4.2 blockade by 4-aminopyridine., Tseng GN., J Pharmacol Exp Ther. November 1, 1996; 279 (2): 865-76.

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