Papers associated with whole organism (and gjb2)

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	Understanding the Role of ATP Release through Connexins Hemichannels during Neurulation., Tovar LM., Int J Mol Sci. January 21, 2023; 24 (3):
	PACmn for improved optogenetic control of intracellular cAMP., Yang S., BMC Biol. October 18, 2021; 19 (1): 227.
	Functional assessment of the "two-hit" model for neurodevelopmental defects in Drosophila and X. laevis., Pizzo L., PLoS Genet. April 5, 2021; 17 (4): e1009112.
	Calmodulin-Cork Model of Gap Junction Channel Gating-One Molecule, Two Mechanisms., Peracchia C., Int J Mol Sci. July 13, 2020; 21 (14):
	Pathogenic FAM83G palmoplantar keratoderma mutations inhibit the PAWS1:CK1α association and attenuate Wnt signalling., Wu KZL., Wellcome Open Res. January 1, 2019; 4 133.
	Concatenation of Human Connexin26 (hCx26) and Human Connexin46 (hCx46) for the Analysis of Heteromeric Gap Junction Hemichannels and Heterotypic Gap Junction Channels., Schadzek P., Int J Mol Sci. September 13, 2018; 19 (9):
	Cell communication across gap junctions: a historical perspective and current developments., Evans WH., Biochem Soc Trans. June 1, 2015; 43 (3): 450-9.
	Aberrant connexin26 hemichannels underlying keratitis-ichthyosis-deafness syndrome are potently inhibited by mefloquine., Levit NA., J Invest Dermatol. April 1, 2015; 135 (4): 1033-1042.
	Long-range gap junctional signaling controls oncogene-mediated tumorigenesis in Xenopus laevis embryos., Chernet BT., Front Physiol. January 19, 2015; 5 519.
	Extracellular gentamicin reduces the activity of connexin hemichannels and interferes with purinergic Ca(2+) signaling in HeLa cells., Figueroa VA., Front Cell Neurosci. May 27, 2014; 8 265.
	Pathological hemichannels associated with human Cx26 mutations causing Keratitis-Ichthyosis-Deafness syndrome., Levit NA., Biochim Biophys Acta. August 1, 2012; 1818 (8): 2014-9.
	Connexin26-mediated transfer of laterality cues in Xenopus., Beyer T., Biol Open. May 15, 2012; 1 (5): 473-81.
	Zebrafish cx30.3: identification and characterization of a gap junction gene highly expressed in the skin., Tao L., Dev Dyn. October 1, 2010; 239 (10): 2627-36.
	Differentially altered Ca2+ regulation and Ca2+ permeability in Cx26 hemichannels formed by the A40V and G45E mutations that cause keratitis ichthyosis deafness syndrome., Sánchez HA., J Gen Physiol. July 1, 2010; 136 (1): 47-62.
	Analysis of four connexin26 mutant gap junctions and hemichannels reveals variations in hexamer stability., Ambrosi C., Biophys J. May 19, 2010; 98 (9): 1809-19.
	Connexin mutations causing skin disease and deafness increase hemichannel activity and cell death when expressed in Xenopus oocytes., Lee JR, Lee JR., J Invest Dermatol. April 1, 2009; 129 (4): 870-8.
	Aberrant hemichannel properties of Cx26 mutations causing skin disease and deafness., Gerido DA., Am J Physiol Cell Physiol. July 1, 2007; 293 (1): C337-45.
	Global analysis of the transcriptional network controlling Xenopus endoderm formation., Sinner D., Development. May 1, 2006; 133 (10): 1955-66.
	An atlas of differential gene expression during early Xenopus embryogenesis., Pollet N., Mech Dev. March 1, 2005; 122 (3): 365-439.
	A novel connexin 26 gene mutation associated with features of the keratitis-ichthyosis-deafness syndrome and the follicular occlusion triad., Montgomery JR., J Am Acad Dermatol. September 1, 2004; 51 (3): 377-82.
	Aberrant gating, but a normal expression pattern, underlies the recessive phenotype of the deafness mutant Connexin26M34T., Skerrett IM., FASEB J. May 1, 2004; 18 (7): 860-2.
	Connexin29 is uniquely distributed within myelinating glial cells of the central and peripheral nervous systems., Altevogt BM., J Neurosci. August 1, 2002; 22 (15): 6458-70.
	trans-dominant inhibition of connexin-43 by mutant connexin-26: implications for dominant connexin disorders affecting epidermal differentiation., Rouan F., J Cell Sci. June 1, 2001; 114 (Pt 11): 2105-13.
	Gap junctions are involved in the early generation of left-right asymmetry., Levin M., Dev Biol. November 1, 1998; 203 (1): 90-105.
	Identification of connexin43 as a functional target for Wnt signalling., van der Heyden MA., J Cell Sci. June 1, 1998; 111 ( Pt 12) 1741-9.
	Molecular cloning and characterization of a new member of the gap junction gene family, connexin-31., Hoh JH., J Biol Chem. April 5, 1991; 266 (10): 6524-31.

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