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XB-ANAT-468
Papers associated with whole organism (and scn4a)
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Loss-of-function mutations in SCN4A cause severe foetal hypokinesia or 'classical' congenital myopathy., Zaharieva IT., Brain. March 1, 2016; 139 (Pt 3): 674-91.  |
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Retention of duplicated ITAM-containing transmembrane signaling subunits in the tetraploid amphibian species Xenopus laevis., Guselnikov SV., Dev Comp Immunol. November 1, 2015; 53 (1): 158-68.  |
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A cation-pi interaction discriminates among sodium channels that are either sensitive or resistant to tetrodotoxin block., Santarelli VP., J Biol Chem. March 16, 2007; 282 (11): 8044-51. |
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Isolation and structure-activity of mu-conotoxin TIIIA, a potent inhibitor of tetrodotoxin-sensitive voltage-gated sodium channels., Lewis RJ., Mol Pharmacol. March 1, 2007; 71 (3): 676-85. |
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A tryptophan residue (W736) in the amino-terminus of the P-segment of domain II is involved in pore formation in Na(v)1.4 voltage-gated sodium channels., Carbonneau E., Pflugers Arch. October 1, 2002; 445 (1): 18-24. |
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