Papers associated with epidermis (and kcnk3)

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	Uricosuric targets of tranilast., Mandal AK., Pharmacol Res Perspect. February 6, 2017; 5 (2): e00291.
	Inhibition of cardiac two-pore-domain K+ (K2P) channels by the antiarrhythmic drug vernakalant--comparison with flecainide., Seyler C., Eur J Pharmacol. February 5, 2014; 724 51-7.
	Class I antiarrhythmic drugs inhibit human cardiac two-pore-domain K(+) (K2 ₂p) channels., Schmidt C., Eur J Pharmacol. December 5, 2013; 721 (1-3): 237-48.
	TASK1 (K(2P)3.1) K(+) channel inhibition by endothelin-1 is mediated through Rho kinase-dependent phosphorylation., Seyler C., Br J Pharmacol. March 1, 2012; 165 (5): 1467-75.
	Carvedilol targets human K2P 3.1 (TASK1) K+ leak channels., Staudacher K., Br J Pharmacol. July 1, 2011; 163 (5): 1099-110.
	In vitro risk assessment of AZD9056 perpetrating a transporter-mediated drug-drug interaction with methotrexate., Elsby R., Eur J Pharm Sci. May 18, 2011; 43 (1-2): 41-9.
	The human cardiac K2P3.1 (TASK-1) potassium leak channel is a molecular target for the class III antiarrhythmic drug amiodarone., Gierten J., Naunyn Schmiedebergs Arch Pharmacol. March 1, 2010; 381 (3): 261-70.
	The flounder organic anion transporter fOat has sequence, function, and substrate specificity similarity to both mammalian Oat1 and Oat3., Aslamkhan AG., Am J Physiol Regul Integr Comp Physiol. December 1, 2006; 291 (6): R1773-80.
	Human organic anion transporter 3 (hOAT3) can operate as an exchanger and mediate secretory urate flux., Bakhiya A., Cell Physiol Biochem. January 1, 2003; 13 (5): 249-56.
	Developmentally regulated expression of organic ion transporters NKT (OAT1), OCT1, NLT (OAT2), and Roct., Pavlova A., Am J Physiol Renal Physiol. April 1, 2000; 278 (4): F635-43.

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