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A novel µ-conopeptide, CnIIIC, exerts potent and preferential inhibition of NaV1.2/1.4 channels and blocks neuronal nicotinic acetylcholine receptors. , Favreau P., Br J Pharmacol. July 1, 2012; 166 (5): 1654-68.
18β-Glycyrrhetinic acid preferentially blocks late Na current generated by ΔKPQ Nav1.5 channels. , Du YM., Acta Pharmacol Sin. June 1, 2012; 33 (6): 752-60.
Induction of vertebrate regeneration by a transient sodium current. , Tseng AS ., J Neurosci. September 29, 2010; 30 (39): 13192-200.
An in vivo tethered toxin approach for the cell-autonomous inactivation of voltage-gated sodium channel currents in nociceptors. , Stürzebecher AS., J Physiol. May 15, 2010; 588 (Pt 10): 1695-707.
Blocking effect of methylflavonolamine on human Na(V)1.5 channels expressed in Xenopus laevis oocytes and on sodium currents in rabbit ventricular myocytes. , Fan XR., Acta Pharmacol Sin. March 1, 2010; 31 (3): 297-306.
An extracellular Cu2+ binding site in the voltage sensor of BK and Shaker potassium channels. , Ma Z., J Gen Physiol. May 1, 2008; 131 (5): 483-502.
Effects of dextrorotatory morphinans on brain Na+ channels expressed in Xenopus oocytes. , Lee JH , Lee JH ., Eur J Pharmacol. June 14, 2007; 564 (1-3): 7-17.
The poison Dart frog's batrachotoxin modulates Nav1.8. , Bosmans F., FEBS Lett. November 5, 2004; 577 (1-2): 245-8.
State-dependent trapping of flecainide in the cardiac sodium channel. , Ramos E., J Physiol. October 1, 2004; 560 (Pt 1): 37-49.