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Validation of TREK1 ion channel activators as an immunomodulatory and neuroprotective strategy in neuroinflammation. , Schroeter CB., Biol Chem. March 28, 2023; 404 (4): 355-375.
Direct action and modulating effect of (+)- and (-)-nicotine on ion channels expressed in trigeminal sensory neurons. , Schreiner BS., Eur J Pharmacol. April 5, 2014; 728 48-58.
The cAMP-binding Popdc proteins have a redundant function in the heart. , Brand T., Biochem Soc Trans. April 1, 2014; 42 (2): 295-301.
Cardiac expression and atrial fibrillation-associated remodeling of K₂p2.1 ( TREK-1) K⁺ channels in a porcine model. , Schmidt C., Life Sci. March 3, 2014; 97 (2): 107-15.
Class I antiarrhythmic drugs inhibit human cardiac two-pore-domain K(+) (K2 ₂p) channels. , Schmidt C., Eur J Pharmacol. December 5, 2013; 721 (1-3): 237-48.
Enhancement of K2P2.1 ( TREK1) background currents expressed in Xenopus oocytes by voltage-gated K+ channel β subunits. , Kisselbach J., Life Sci. October 5, 2012; 91 (11-12): 377-383.
TREK-1 isoforms generated by alternative translation initiation display different susceptibility to the antidepressant fluoxetine. , Eckert M., Neuropharmacology. January 1, 2011; 61 (5-6): 918-23.
Differential effects of volatile and intravenous anesthetics on the activity of human TASK-1. , Putzke C., Am J Physiol Cell Physiol. October 1, 2007; 293 (4): C1319-26.
Cloning, functional expression and brain localization of a novel unconventional outward rectifier K+ channel. , Fink M., EMBO J. December 16, 1996; 15 (24): 6854-62.