Papers associated with right (and kcnj10)

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	Advancements in the use of xenopus oocytes for modelling neurological disease for novel drug discovery., O'Connor EC., Expert Opin Drug Discov. February 1, 2024; 19 (2): 173-187.
	Identification of a unique endoplasmic retention motif in the Xenopus GIRK5 channel and its contribution to oocyte maturation., Rangel-Garcia CI., FEBS Open Bio. April 1, 2021; 11 (4): 1093-1108.
	Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy., Sicca F., Sci Rep. September 28, 2016; 6 34325.
	TRPV4 and AQP4 Channels Synergistically Regulate Cell Volume and Calcium Homeostasis in Retinal Müller Glia., Jo AO., J Neurosci. September 30, 2015; 35 (39): 13525-37.
	Transmembrane voltage potential is an essential cellular parameter for the detection and control of tumor development in a Xenopus model., Chernet BT., Dis Model Mech. May 1, 2013; 6 (3): 595-607.
	Mutations of KCNJ10 together with mutations of SLC26A4 cause digenic nonsyndromic hearing loss associated with enlarged vestibular aqueduct syndrome., Yang T., Am J Hum Genet. May 1, 2009; 84 (5): 651-7.
	H,K-ATPase protein localization and Kir4.1 function reveal concordance of three axes during early determination of left-right asymmetry., Aw S., Mech Dev. January 1, 2008; 125 (3-4): 353-72.
	Asymmetries in H+/K+-ATPase and cell membrane potentials comprise a very early step in left-right patterning., Levin M., Cell. October 4, 2002; 111 (1): 77-89.
	Modulation of the heteromeric Kir4.1-Kir5.1 channels by P(CO(2)) at physiological levels., Cui N., J Cell Physiol. November 1, 2001; 189 (2): 229-36.

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