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Summary Anatomy Item Literature (4079) Expression Attributions Wiki
XB-ANAT-3714

Papers associated with right (and kcnj11)

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A loss-of-function mutation in KCNJ11 causing sulfonylurea-sensitive diabetes in early adult life., Vedovato N., Diabetologia. May 1, 2024; 67 (5): 940-951.          

Identification of a unique endoplasmic retention motif in the Xenopus GIRK5 channel and its contribution to oocyte maturation., Rangel-Garcia CI., FEBS Open Bio. April 1, 2021; 11 (4): 1093-1108.            

Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome., Adams DS., J Physiol. June 15, 2016; 594 (12): 3245-70.                              

Successful transfer to sulfonylureas in KCNJ11 neonatal diabetes is determined by the mutation and duration of diabetes., Babiker T., Diabetologia. June 1, 2016; 59 (6): 1162-6.    

Sensitivity of KATP channels to cellular metabolic disorders and the underlying structural basis., Li CG., Acta Pharmacol Sin. January 1, 2016; 37 (1): 134-42.        

Kir6.2 activation by sulfonylurea receptors: a different mechanism of action for SUR1 and SUR2A subunits via the same residues., Principalli MA., Physiol Rep. September 1, 2015; 3 (9):         

Activation of the K(ATP) channel by Mg-nucleotide interaction with SUR1., Proks P., J Gen Physiol. October 1, 2010; 136 (4): 389-405.                    

How ATP inhibits the open K(ATP) channel., Craig TJ., J Gen Physiol. July 1, 2008; 132 (1): 131-44.            

Ligand-dependent linkage of the ATP site to inhibition gate closure in the KATP channel., Li L., J Gen Physiol. September 1, 2005; 126 (3): 285-99.                  

Molecular basis of inward rectification: polyamine interaction sites located by combined channel and ligand mutagenesis., Kurata HT., J Gen Physiol. November 1, 2004; 124 (5): 541-54.                

K(ATP) channel activity is required for hatching in Xenopus embryos., Cheng SM., Dev Dyn. December 1, 2002; 225 (4): 588-91.        

Open state destabilization by ATP occupancy is mechanism speeding burst exit underlying KATP channel inhibition by ATP., Li L., J Gen Physiol. January 1, 2002; 119 (1): 105-16.                    

Mutations within the P-loop of Kir6.2 modulate the intraburst kinetics of the ATP-sensitive potassium channel., Proks P., J Gen Physiol. October 1, 2001; 118 (4): 341-53.                    

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