Papers associated with right (and cftr)

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	VX-770-mediated potentiation of numerous human CFTR disease mutants is influenced by phosphorylation level., Cui G., Sci Rep. September 17, 2019; 9 (1): 13460.
	Asymmetry of movements in CFTR's two ATP sites during pore opening serves their distinct functions., Sorum B., Elife. September 25, 2017; 6
	Hydrogen sulfide stimulates CFTR in Xenopus oocytes by activation of the cAMP/PKA signalling axis., Perniss A., Sci Rep. June 14, 2017; 7 (1): 3517.
	Cysteine accessibility probes timing and extent of NBD separation along the dimer interface in gating CFTR channels., Chaves LA., J Gen Physiol. April 1, 2015; 145 (4): 261-83.
	Structure-activity analysis of a CFTR channel potentiator: Distinct molecular parts underlie dual gating effects., Csanády L., J Gen Physiol. October 1, 2014; 144 (4): 321-36.
	Three charged amino acids in extracellular loop 1 are involved in maintaining the outer pore architecture of CFTR., Cui G., J Gen Physiol. August 1, 2014; 144 (2): 159-79.
	Comparative expression analysis of cysteine-rich intestinal protein family members crip1, 2 and 3 during Xenopus laevis embryogenesis., Hempel A., Int J Dev Biol. January 1, 2014; 58 (10-12): 841-9.
	Human trace amine-associated receptor TAAR5 can be activated by trimethylamine., Wallrabenstein I., PLoS One. January 1, 2013; 8 (2): e54950.
	Mutant cycles at CFTR's non-canonical ATP-binding site support little interface separation during gating., Szollosi A., J Gen Physiol. June 1, 2011; 137 (6): 549-62.
	Involvement of F1296 and N1303 of CFTR in induced-fit conformational change in response to ATP binding at NBD2., Szollosi A., J Gen Physiol. October 1, 2010; 136 (4): 407-23.
	The muscle chloride channel ClC-1 is not directly regulated by intracellular ATP., Zifarelli G., J Gen Physiol. February 1, 2008; 131 (2): 109-16.
	Preferential phosphorylation of R-domain Serine 768 dampens activation of CFTR channels by PKA., Csanády L., J Gen Physiol. February 1, 2005; 125 (2): 171-86.
	Functional roles of nonconserved structural segments in CFTR's NH2-terminal nucleotide binding domain., Csanády L., J Gen Physiol. January 1, 2005; 125 (1): 43-55.
	Prolonged nonhydrolytic interaction of nucleotide with CFTR's NH2-terminal nucleotide binding domain and its role in channel gating., Basso C., J Gen Physiol. September 1, 2003; 122 (3): 333-48.
	On the mechanism of MgATP-dependent gating of CFTR Cl- channels., Vergani P., J Gen Physiol. January 1, 2003; 121 (1): 17-36.
	Severed channels probe regulation of gating of cystic fibrosis transmembrane conductance regulator by its cytoplasmic domains., Csanády L., J Gen Physiol. September 1, 2000; 116 (3): 477-500.
	Severed molecules functionally define the boundaries of the cystic fibrosis transmembrane conductance regulator's NH(2)-terminal nucleotide binding domain., Chan KW., J Gen Physiol. August 1, 2000; 116 (2): 163-80.
	Heterologous facilitation of G protein-activated K(+) channels by beta-adrenergic stimulation via cAMP-dependent protein kinase., Müllner C., J Gen Physiol. May 1, 2000; 115 (5): 547-58.
	Dual effects of ADP and adenylylimidodiphosphate on CFTR channel kinetics show binding to two different nucleotide binding sites., Weinreich F., J Gen Physiol. July 1, 1999; 114 (1): 55-70.

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