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Summary Anatomy Item Literature (4079) Expression Attributions Wiki
XB-ANAT-3714

Papers associated with right (and pou5f3)

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A maternal dorsoventral prepattern revealed by an asymmetric distribution of ventralizing molecules before fertilization in Xenopus laevis., Castro Colabianchi AM., Front Cell Dev Biol. January 1, 2024; 12 1365705.                


Hybridization led to a rewired pluripotency network in the allotetraploid Xenopus laevis., Phelps WA., Elife. October 3, 2023; 12                             


Membrane potential drives the exit from pluripotency and cell fate commitment via calcium and mTOR., Sempou E., Nat Commun. November 5, 2022; 13 (1): 6681.                                            


The DNA-to-cytoplasm ratio broadly activates zygotic gene expression in Xenopus., Jukam D., Curr Biol. October 11, 2021; 31 (19): 4269-4281.e8.                          


Combinatorial transcription factor activities on open chromatin induce embryonic heterogeneity in vertebrates., Bright AR., EMBO J. May 3, 2021; 40 (9): e104913.                        


Chromatin accessibility and histone acetylation in the regulation of competence in early development., Esmaeili M., Dev Biol. June 1, 2020; 462 (1): 20-35.                


Maternal pluripotency factors initiate extensive chromatin remodelling to predefine first response to inductive signals., Gentsch GE., Nat Commun. September 19, 2019; 10 (1): 4269.                                        


A Critical E-box in Barhl1 3' Enhancer Is Essential for Auditory Hair Cell Differentiation., Hou K., Cells. May 15, 2019; 8 (5):               


Leukemia inhibitory factor signaling in Xenopus embryo: Insights from gain of function analysis and dominant negative mutant of the receptor., Jalvy S., Dev Biol. March 15, 2019; 447 (2): 200-213.                                  


Serine Threonine Kinase Receptor-Associated Protein Deficiency Impairs Mouse Embryonic Stem Cells Lineage Commitment Through CYP26A1-Mediated Retinoic Acid Homeostasis., Jin L., Stem Cells. September 1, 2018; 36 (9): 1368-1379.                      


Chromatin Accessibility Impacts Transcriptional Reprogramming in Oocytes., Miyamoto K., Cell Rep. July 10, 2018; 24 (2): 304-311.          


sall1 and sall4 repress pou5f3 family expression to allow neural patterning, differentiation, and morphogenesis in Xenopus laevis., Exner CRT., Dev Biol. May 1, 2017; 425 (1): 33-43.                                    


Pou5f3.2-induced proliferative state of embryonic cells during gastrulation of Xenopus laevis embryo., Nishitani E., Dev Growth Differ. December 1, 2015; 57 (9): 591-600.              


Genome-wide view of TGFβ/Foxh1 regulation of the early mesendoderm program., Chiu WT., Development. December 1, 2014; 141 (23): 4537-47.                                  


Spalt-like 4 promotes posterior neural fates via repression of pou5f3 family members in Xenopus., Young JJ., Development. April 1, 2014; 141 (8): 1683-93.                                                                


A conserved Oct4/POUV-dependent network links adhesion and migration to progenitor maintenance., Livigni A., Curr Biol. November 18, 2013; 23 (22): 2233-2244.                                    


TBX3 Directs Cell-Fate Decision toward Mesendoderm., Weidgang CE., Stem Cell Reports. August 29, 2013; 1 (3): 248-65.                


MRAS GTPase is a novel stemness marker that impacts mouse embryonic stem cell plasticity and Xenopus embryonic cell fate., Mathieu ME., Development. August 1, 2013; 140 (16): 3311-22.              


Expression of pluripotency factors in larval epithelia of the frog Xenopus: evidence for the presence of cornea epithelial stem cells., Perry KJ., Dev Biol. February 15, 2013; 374 (2): 281-94.                


Pou-V factor Oct25 regulates early morphogenesis in Xenopus laevis., Julier A., Dev Growth Differ. September 1, 2012; 54 (7): 702-16.              


Ventx factors function as Nanog-like guardians of developmental potential in Xenopus., Scerbo P., PLoS One. January 1, 2012; 7 (5): e36855.              


Histone variant macroH2A confers resistance to nuclear reprogramming., Pasque V., EMBO J. May 6, 2011; 30 (12): 2373-87.                


Gadd45a and Gadd45g regulate neural development and exit from pluripotency in Xenopus., Kaufmann LT., Mech Dev. January 1, 2011; 128 (7-10): 401-11.                      


Gemcitabine functions epigenetically by inhibiting repair mediated DNA demethylation., Schäfer A., PLoS One. November 19, 2010; 5 (11): e14060.            


Characterization of somatic cell nuclear reprogramming by oocytes in which a linker histone is required for pluripotency gene reactivation., Jullien J., Proc Natl Acad Sci U S A. March 23, 2010; 107 (12): 5483-8.        


Histone H3 lysine 4 methylation is associated with the transcriptional reprogramming efficiency of somatic nuclei by oocytes., Murata K., Epigenetics Chromatin. February 4, 2010; 3 (1): 4.              


The Oct4 homologue PouV and Nanog regulate pluripotency in chicken embryonic stem cells., Lavial F., Development. October 1, 2007; 134 (19): 3549-63.      


Conserved roles for Oct4 homologues in maintaining multipotency during early vertebrate development., Morrison GM., Development. May 1, 2006; 133 (10): 2011-22.                


The murine ortholog of notchless, a direct regulator of the notch pathway in Drosophila melanogaster, is essential for survival of inner cell mass cells., Cormier S., Mol Cell Biol. May 1, 2006; 26 (9): 3541-9.              


GDF3, a BMP inhibitor, regulates cell fate in stem cells and early embryos., Levine AJ., Development. January 1, 2006; 133 (2): 209-16.            


Gene expression screening in Xenopus identifies molecular pathways, predicts gene function and provides a global view of embryonic patterning., Gawantka V., Mech Dev. October 1, 1998; 77 (2): 95-141.                                                            


Localized expression of a Xenopus POU gene depends on cell-autonomous transcriptional activation and induction-dependent inactivation., Frank D., Development. June 1, 1992; 115 (2): 439-48.            

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