Papers associated with right (and xrcc1)

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	DNA ligase III acts as a DNA strand break sensor in the cellular orchestration of DNA strand break repair., Abdou I., Nucleic Acids Res. January 1, 2015; 43 (2): 875-92.
	Damage response of XRCC1 at sites of DNA single strand breaks is regulated by phosphorylation and ubiquitylation after degradation of poly(ADP-ribose)., Wei L., J Cell Sci. October 1, 2013; 126 (Pt 19): 4414-23.
	Functional redundancy between DNA ligases I and III in DNA replication in vertebrate cells., Arakawa H., Nucleic Acids Res. March 1, 2012; 40 (6): 2599-610.
	Cardiac neural crest is dispensable for outflow tract septation in Xenopus., Lee YH., Development. May 1, 2011; 138 (10): 2025-34.
	XRCC1 interacts with the p58 subunit of DNA Pol alpha-primase and may coordinate DNA repair and replication during S phase., Lévy N., Nucleic Acids Res. June 1, 2009; 37 (10): 3177-88.
	Aprataxin, causative gene product for EAOH/AOA1, repairs DNA single-strand breaks with damaged 3'-phosphate and 3'-phosphoglycolate ends., Takahashi T., Nucleic Acids Res. January 1, 2007; 35 (11): 3797-809.
	ATM mediates oxidative stress-induced dephosphorylation of DNA ligase IIIalpha., Dong Z., Nucleic Acids Res. January 1, 2006; 34 (20): 5721-279.
	Translocation of XRCC1 and DNA ligase IIIalpha from centrosomes to chromosomes in response to DNA damage in mitotic human cells., Okano S., Nucleic Acids Res. January 12, 2005; 33 (1): 422-9.

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