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Association of A Novel Splice Site Mutation in P/Q-Type Calcium Channels with Childhood Epilepsy and Late-Onset Slowly Progressive Non-Episodic Cerebellar Ataxia. , Stendel C., Int J Mol Sci. May 27, 2020; 21 (11):
Sigma-1 Receptor Plays a Negative Modulation on N-type Calcium Channel. , Zhang K., Front Pharmacol. May 26, 2017; 8 302.
Honeybee locomotion is impaired by Am- CaV3 low voltage-activated Ca2+ channel antagonist. , Rousset M., Sci Rep. February 1, 2017; 7 41782.
Less is More: Design of a Highly Stable Disulfide-Deleted Mutant of Analgesic Cyclic α-Conotoxin Vc1.1. , Yu R., Sci Rep. August 20, 2015; 5 13264.
A Single Amino Acid Deletion (ΔF1502) in the S6 Segment of CaV2.1 Domain III Associated with Congenital Ataxia Increases Channel Activity and Promotes Ca2+ Influx. , Bahamonde MI., PLoS One. January 1, 2015; 10 (12): e0146035.
A post-burst after depolarization is mediated by group i metabotropic glutamate receptor-dependent upregulation of Ca(v)2.3 R-type calcium channels in CA1 pyramidal neurons. , Park JY ., PLoS Biol. November 16, 2010; 8 (11): e1000534.
N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2. , Page KM., J Biol Chem. January 8, 2010; 285 (2): 835-44.
Disruption of the IS6-AID linker affects voltage-gated calcium channel inactivation and facilitation. , Findeisen F., J Gen Physiol. March 1, 2009; 133 (3): 327-43.
Overexpressed Ca(v)beta3 inhibits N-type ( Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation. , Yasuda T., J Gen Physiol. April 1, 2004; 123 (4): 401-16.