Papers associated with regenerating tissue (and mink1)

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	Mink1 regulates β-catenin-independent Wnt signaling via Prickle phosphorylation., Daulat AM., Mol Cell Biol. January 1, 2012; 32 (1): 173-85.
	Dynamic regulation of Emi2 by Emi2-bound Cdk1/Plk1/CK1 and PP2A-B56 in meiotic arrest of Xenopus eggs., Isoda M., Dev Cell. September 13, 2011; 21 (3): 506-19.
	Acute effects of dronedarone on both components of the cardiac delayed rectifier K+ current, HERG and KvLQT1/minK potassium channels., Thomas D., Br J Pharmacol. November 1, 2003; 140 (5): 996-1002.
	Clinical and electrophysiological characterization of a novel mutation (F193L) in the KCNQ1 gene associated with long QT syndrome., Yamaguchi M., Clin Sci (Lond). April 1, 2003; 104 (4): 377-82.
	Inhibitory effects of volatile anesthetics on currents produced on heterologous expression of KvLQT1 and minK in Xenopus oocytes., Chen X., Vascul Pharmacol. July 1, 2002; 39 (1-2): 33-8.
	MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
	Increase of the single-channel conductance of KvLQT1 potassium channels induced by the association with minK., Pusch M., Pflugers Arch. December 1, 1998; 437 (1): 172-4.
	Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits., Tristani-Firouzi M., J Physiol. July 1, 1998; 510 ( Pt 1) 37-45.
	Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.

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