Papers associated with muscle (and nav1)

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	Extracellular cysteine disulfide bond break at Cys122 disrupts PIP 2 -dependent Kir2.1 channel function and leads to arrhythmias in Andersen-Tawil Syndrome., Cruz FM., bioRxiv. June 8, 2023;
	Differential regulation of cardiac sodium channels by intracellular fibroblast growth factors., Angsutararux P., J Gen Physiol. May 1, 2023; 155 (5):
	The G213D variant in Nav1.5 alters sodium current and causes an arrhythmogenic phenotype resulting in a multifocal ectopic Purkinje-related premature contraction phenotype in human-induced pluripotent stem cell-derived cardiomyocytes., Calloe K., Europace. December 9, 2022; 24 (12): 2015-2027.
	Identification of SCN5a p.C335R Variant in a Large Family with Dilated Cardiomyopathy and Conduction Disease., Sedaghat-Hamedani F., Int J Mol Sci. November 30, 2021; 22 (23):
	Heterologous functional expression of ascidian Nav1 channels and close relationship with the evolutionary ancestor of vertebrate Nav channels., Kawai T., J Biol Chem. January 1, 2021; 296 100783.
	Action potentials in Xenopus oocytes triggered by blue light., Walther F., J Gen Physiol. May 4, 2020; 152 (5):
	Polyunsaturated fatty acid analogues differentially affect cardiac NaV, CaV, and KV channels through unique mechanisms., Bohannon BM., Elife. March 24, 2020; 9
	Myasthenic congenital myopathy from recessive mutations at a single residue in NaV1.4., Elia N., Neurology. March 26, 2019; 92 (13): e1405-e1415.
	Molecular charge associated with antiarrhythmic actions in a series of amino-2-cyclohexyl ester derivatives., Pugsley MK., Eur J Pharmacol. February 5, 2019; 844 241-252.
	Spider toxin inhibits gating pore currents underlying periodic paralysis., Männikkö R., Proc Natl Acad Sci U S A. April 24, 2018; 115 (17): 4495-4500.
	Mapping of voltage sensor positions in resting and inactivated mammalian sodium channels by LRET., Kubota T., Proc Natl Acad Sci U S A. March 7, 2017; 114 (10): E1857-E1865.
	Fast-onset lidocaine block of rat NaV1.4 channels suggests involvement of a second high-affinity open state., Gingrich KJ., Biochim Biophys Acta. June 1, 2016; 1858 (6): 1175-88.
	Splicing misregulation of SCN5A contributes to cardiac-conduction delay and heart arrhythmia in myotonic dystrophy., Freyermuth F., Nat Commun. April 11, 2016; 7 11067.
	Loss-of-function mutations in SCN4A cause severe foetal hypokinesia or 'classical' congenital myopathy., Zaharieva IT., Brain. March 1, 2016; 139 (Pt 3): 674-91.
	Bioelectric memory: modeling resting potential bistability in amphibian embryos and mammalian cells., Law R., Plant Physiol. October 15, 2015; 12 22.
	Voltage-dependent blockade by bupivacaine of cardiac sodium channels expressed in Xenopus oocytes., Zhang H., Neurosci Bull. August 1, 2014; 30 (4): 697-710.
	Disrupted coupling of gating charge displacement to Na+ current activation for DIIS4 mutations in hypokalemic periodic paralysis., Mi W., J Gen Physiol. August 1, 2014; 144 (2): 137-45.
	Asymmetric functional contributions of acidic and aromatic side chains in sodium channel voltage-sensor domains., Pless SA., J Gen Physiol. May 1, 2014; 143 (5): 645-56.
	Voltage-sensor movements describe slow inactivation of voltage-gated sodium channels II: a periodic paralysis mutation in Na(V)1.4 (L689I)., Silva JR., J Gen Physiol. March 1, 2013; 141 (3): 323-34.
	Voltage-sensor movements describe slow inactivation of voltage-gated sodium channels I: wild-type skeletal muscle Na(V)1.4., Silva JR., J Gen Physiol. March 1, 2013; 141 (3): 309-21.
	A novel µ-conopeptide, CnIIIC, exerts potent and preferential inhibition of NaV1.2/1.4 channels and blocks neuronal nicotinic acetylcholine receptors., Favreau P., Br J Pharmacol. July 1, 2012; 166 (5): 1654-68.
	18β-Glycyrrhetinic acid preferentially blocks late Na current generated by ΔKPQ Nav1.5 channels., Du YM., Acta Pharmacol Sin. June 1, 2012; 33 (6): 752-60.
	A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype., Gosselin-Badaroudine P., PLoS One. January 1, 2012; 7 (5): e38331.
	Molecular mechanism of allosteric modification of voltage-dependent sodium channels by local anesthetics., Arcisio-Miranda M., J Gen Physiol. November 1, 2010; 136 (5): 541-54.
	Blocking effect of methylflavonolamine on human Na(V)1.5 channels expressed in Xenopus laevis oocytes and on sodium currents in rabbit ventricular myocytes., Fan XR., Acta Pharmacol Sin. March 1, 2010; 31 (3): 297-306.
	Electric currents in Xenopus tadpole tail regeneration., Reid B., Dev Biol. November 1, 2009; 335 (1): 198-207.
	Slow inactivation of the NaV1.4 sodium channel in mammalian cells is impeded by co-expression of the beta1 subunit., Webb J., Pflugers Arch. April 1, 2009; 457 (6): 1253-63.
	Correlations between clinical and physiological consequences of the novel mutation R878C in a highly conserved pore residue in the cardiac Na+ channel., Zhang Y., Acta Physiol (Oxf). December 1, 2008; 194 (4): 311-23.
	Alpha-scorpion toxin impairs a conformational change that leads to fast inactivation of muscle sodium channels., Campos FV., J Gen Physiol. August 1, 2008; 132 (2): 251-63.
	Involvement of batrachotoxin binding sites in ginsenoside-mediated voltage-gated Na+ channel regulation., Lee JH., Brain Res. April 8, 2008; 1203 61-7.
	A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore., Struyk AF., J Gen Physiol. July 1, 2007; 130 (1): 11-20.
	Solution structure of Jingzhaotoxin-III, a peptide toxin inhibiting both Nav1.5 and Kv2.1 channels., Liao Z., Toxicon. July 1, 2007; 50 (1): 135-43.
	A cation-pi interaction discriminates among sodium channels that are either sensitive or resistant to tetrodotoxin block., Santarelli VP., J Biol Chem. March 16, 2007; 282 (11): 8044-51.
	Isolation and structure-activity of mu-conotoxin TIIIA, a potent inhibitor of tetrodotoxin-sensitive voltage-gated sodium channels., Lewis RJ., Mol Pharmacol. March 1, 2007; 71 (3): 676-85.
	A mutation in the local anaesthetic binding site abolishes toluene effects in sodium channels., Gauthereau MY., Eur J Pharmacol. December 28, 2005; 528 (1-3): 17-26.
	Occurrence of a tetrodotoxin-sensitive calcium current in rat ventricular myocytes after long-term myocardial infarction., Alvarez JL., Cardiovasc Res. September 1, 2004; 63 (4): 653-61.
	Molecular cloning and functional expression of the human sodium channel beta1B subunit, a novel splicing variant of the beta1 subunit., Qin N., Eur J Biochem. December 1, 2003; 270 (23): 4762-70.
	Inhibition of cardiac sodium currents by toluene exposure., Cruz SL., Br J Pharmacol. October 1, 2003; 140 (4): 653-60.

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