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Summary Anatomy Item Literature (3673) Expression Attributions Wiki
XB-ANAT-490

Papers associated with tail (and avp)

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Impaired negative feedback and death following acute stress in glucocorticoid receptor knockout Xenopus tropicalis tadpoles., Paul B., Gen Comp Endocrinol. September 15, 2022; 326 114072.      


fus/TLS orchestrates splicing of developmental regulators during gastrulation., Dichmann DS., Genes Dev. June 15, 2012; 26 (12): 1351-63.                        


Aquaporin-2: new mutations responsible for autosomal-recessive nephrogenic diabetes insipidus-update and epidemiology., Bichet DG., Clin Kidney J. June 1, 2012; 5 (3): 195-202.          


Role of multiple phosphorylation sites in the COOH-terminal tail of aquaporin-2 for water transport: evidence against channel gating., Moeller HB., Am J Physiol Renal Physiol. March 1, 2009; 296 (3): F649-57.


Characterization of the neurohypophysial hormone gene loci in elephant shark and the Japanese lamprey: origin of the vertebrate neurohypophysial hormone genes., Gwee PC., BMC Evol Biol. February 26, 2009; 9 47.              


Sequence and organization of coelacanth neurohypophysial hormone genes: evolutionary history of the vertebrate neurohypophysial hormone gene locus., Gwee PC., BMC Evol Biol. February 8, 2008; 8 93.            


Neuronal, neurohormonal, and autocrine control of Xenopus melanotrope cell activity., Roubos EW., Ann N Y Acad Sci. April 1, 2005; 1040 172-83.


A novel mechanism in recessive nephrogenic diabetes insipidus: wild-type aquaporin-2 rescues the apical membrane expression of intracellularly retained AQP2-P262L., de Mattia F., Hum Mol Genet. December 15, 2004; 13 (24): 3045-56.


Cloning of Octopus cephalotocin receptor, a member of the oxytocin/vasopressin superfamily., Kanda A., J Endocrinol. November 1, 2003; 179 (2): 281-91.


Three families with autosomal dominant nephrogenic diabetes insipidus caused by aquaporin-2 mutations in the C-terminus., Kuwahara M., Am J Hum Genet. October 1, 2001; 69 (4): 738-48.


Injection of rat hepatocyte poly(A)+ RNA to Xenopus laevis oocytes leads to expression of a constitutively-active divalent cation channel distinguishable from endogenous receptor-activated channels., Auld AM., Cell Calcium. May 1, 1996; 19 (5): 439-52.


Adenosine stimulation of Na+ transport is mediated by an A1 receptor and a [Ca2+]i-dependent mechanism., Hayslett JP., Kidney Int. June 1, 1995; 47 (6): 1576-84.


[Recent progress in molecular biology of inherited tubular transport abnormalities]., Indo Y., Nihon Rinsho. December 1, 1992; 50 (12): 3086-92.


Mechanisms of subcellular cytosolic Ca2+ signaling evoked by stimulation of the vasopressin V1a receptor., Nathanson MH., J Biol Chem. November 15, 1992; 267 (32): 23282-9.


Impact of altered protein structures on the intracellular traffic of a mutated vasopressin precursor from Brattleboro rats., Schmale H., Eur J Biochem. July 1, 1989; 182 (3): 621-7.

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