Papers associated with tail (and cftr)

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	Estimating the true stability of the prehydrolytic outward-facing state in an ABC protein., Simon MA., Elife. October 2, 2023; 12
	UXT chaperone prevents proteotoxicity by acting as an autophagy adaptor for p62-dependent aggrephagy., Yoon MJ., Nat Commun. March 29, 2021; 12 (1): 1955.
	Structure-activity analysis of a CFTR channel potentiator: Distinct molecular parts underlie dual gating effects., Csanády L., J Gen Physiol. October 1, 2014; 144 (4): 321-36.
	Comparative expression analysis of cysteine-rich intestinal protein family members crip1, 2 and 3 during Xenopus laevis embryogenesis., Hempel A., Int J Dev Biol. January 1, 2014; 58 (10-12): 841-9.
	Conformational changes in the catalytically inactive nucleotide-binding site of CFTR., Csanády L., J Gen Physiol. July 1, 2013; 142 (1): 61-73.
	Involvement of F1296 and N1303 of CFTR in induced-fit conformational change in response to ATP binding at NBD2., Szollosi A., J Gen Physiol. October 1, 2010; 136 (4): 407-23.
	Strict coupling between CFTR's catalytic cycle and gating of its Cl- ion pore revealed by distributions of open channel burst durations., Csanády L., Proc Natl Acad Sci U S A. January 19, 2010; 107 (3): 1241-6.
	AMP-activated protein kinase phosphorylation of the R domain inhibits PKA stimulation of CFTR., King JD., Am J Physiol Cell Physiol. July 1, 2009; 297 (1): C94-101.
	Imaging CFTR in its native environment., Schillers H., Pflugers Arch. April 1, 2008; 456 (1): 163-77.
	The muscle chloride channel ClC-1 is not directly regulated by intracellular ATP., Zifarelli G., J Gen Physiol. February 1, 2008; 131 (2): 109-16.
	In vivo phosphorylation of CFTR promotes formation of a nucleotide-binding domain heterodimer., Mense M., EMBO J. October 18, 2006; 25 (20): 4728-39.
	Control of epithelial ion transport by Cl- and PDZ proteins., Schreiber R., J Membr Biol. May 15, 2004; 199 (2): 85-98.
	Imaging CFTR: a tail to tail dimer with a central pore., Schillers H., Cell Physiol Biochem. January 1, 2004; 14 (1-2): 1-10.
	[Regulation of the drug-sensitivity of anion channels via phosphorylation]., Yamazaki J., Nihon Yakurigaku Zasshi. November 1, 2003; 122 Suppl 67P-70P.
	Acute regulation of the SLC26A3 congenital chloride diarrhoea anion exchanger (DRA) expressed in Xenopus oocytes., Chernova MN., J Physiol. May 15, 2003; 549 (Pt 1): 3-19.
	The interaction between syntaxin 1A and cystic fibrosis transmembrane conductance regulator Cl- channels is mechanistically distinct from syntaxin 1A-SNARE interactions., Ganeshan R., J Biol Chem. January 31, 2003; 278 (5): 2876-85.
	A cluster of negative charges at the amino terminal tail of CFTR regulates ATP-dependent channel gating., Fu J., J Physiol. October 15, 2001; 536 (Pt 2): 459-70.
	Cysteine substitutions reveal dual functions of the amino-terminal tail in cystic fibrosis transmembrane conductance regulator channel gating., Fu J., J Biol Chem. September 21, 2001; 276 (38): 35660-8.
	Anion permeation in Ca(2+)-activated Cl(-) channels., Qu Z., J Gen Physiol. December 1, 2000; 116 (6): 825-44.
	Effect of genistein on native epithelial tissue from normal individuals and CF patients and on ion channels expressed in Xenopus oocytes., Mall M., Br J Pharmacol. August 1, 2000; 130 (8): 1884-92.
	CFTR chloride channel regulation by an interdomain interaction., Naren AP., Science. October 15, 1999; 286 (5439): 544-8.
	Capacitance measurements reveal different pathways for the activation of CFTR., Weber WM., Pflugers Arch. September 1, 1999; 438 (4): 561-9.
	Syntaxin 1A inhibits CFTR chloride channels by means of domain-specific protein-protein interactions., Naren AP., Proc Natl Acad Sci U S A. September 1, 1998; 95 (18): 10972-7.
	cAMP-stimulated ion currents in Xenopus oocytes expressing CFTR cRNA., Cunningham SA., Am J Physiol. March 1, 1992; 262 (3 Pt 1): C783-8.

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