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Functional Characterization of a Spectrum of Novel Romano-Ward Syndrome KCNQ1 Variants. , Rinné S., Int J Mol Sci. January 10, 2023; 24 (2):
Pharmacological rescue of specific long QT variants of KCNQ1/KCNE1 channels. , Zou X., Front Physiol. January 1, 2022; 13 902224.
Bioelectric signaling: Reprogrammable circuits underlying embryogenesis, regeneration, and cancer. , Levin M ., Cell. April 15, 2021;
Molecular Mechanism of Autosomal Recessive Long QT-Syndrome 1 without Deafness. , Oertli A., Int J Mol Sci. January 23, 2021; 22 (3):
Eag Domains Regulate LQT Mutant hERG Channels in Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes. , Liu QN., PLoS One. April 22, 2015; 10 (4): e0123951.
High incidence of functional ion-channel abnormalities in a consecutive Long QT cohort with novel missense genetic variants of unknown significance. , Steffensen AB., Sci Rep. January 12, 2015; 5 10009.
Domain-domain interactions determine the gating, permeation, pharmacology, and subunit modulation of the IKs ion channel. , Zaydman MA., Elife. March 12, 2014; 3 e03606.
Extracellular potassium inhibits Kv7.1 potassium channels by stabilizing an inactivated state. , Larsen AP., Biophys J. August 17, 2011; 101 (4): 818-27.
The acrylamide (S)-2 as a positive and negative modulator of Kv7 channels expressed in Xenopus laevis oocytes. , Blom SM., PLoS One. December 11, 2009; 4 (12): e8251.
S1 constrains S4 in the voltage sensor domain of Kv7.1 K+ channels. , Haitin Y., PLoS One. April 9, 2008; 3 (4): e1935.
KCNE1 constrains the voltage sensor of Kv7.1 K+ channels. , Shamgar L., PLoS One. April 9, 2008; 3 (4): e1943.
Acute effects of dronedarone on both components of the cardiac delayed rectifier K+ current, HERG and KvLQT1/ minK potassium channels. , Thomas D., Br J Pharmacol. November 1, 2003; 140 (5): 996-1002.
Inhibitory effects of volatile anesthetics on currents produced on heterologous expression of KvLQT1 and minK in Xenopus oocytes. , Chen X., Vascul Pharmacol. July 1, 2002; 39 (1-2): 33-8.
MinK subdomains that mediate modulation of and association with KvLQT1. , Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
Molecular and functional characterization of the small Ca(2+)-regulated K+ channel ( rSK4) of colonic crypts. , Warth R., Pflugers Arch. September 1, 1999; 438 (4): 437-44.
Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome. , Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
Increase of the single-channel conductance of KvLQT1 potassium channels induced by the association with minK. , Pusch M., Pflugers Arch. December 1, 1998; 437 (1): 172-4.
Activation and inactivation of homomeric KvLQT1 potassium channels. , Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.
Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel ( minK) subunits. , Tristani-Firouzi M., J Physiol. July 1, 1998; 510 ( Pt 1) 37-45.
Block by propofol and thiopentone of the min K current ( IsK) expressed in Xenopus oocytes. , Heath BM., Naunyn Schmiedebergs Arch Pharmacol. September 1, 1997; 356 (3): 404-9.