Papers associated with tail (and hcn1)

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	External Cd2+ and protons activate the hyperpolarization-gated K+ channel KAT1 at the voltage sensor., Zhou Y., J Gen Physiol. January 4, 2021; 153 (1):
	Bipolar switching by HCN voltage sensor underlies hyperpolarization activation., Cowgill J., Proc Natl Acad Sci U S A. January 8, 2019; 116 (2): 670-678.
	The VAMP-associated protein VAPB is required for cardiac and neuronal pacemaker channel function., Silbernagel N., FASEB J. November 1, 2018; 32 (11): 6159-6173.
	Minimal molecular determinants of isoform-specific differences in efficacy in the HCN channel family., Alvarez-Baron CP., J Gen Physiol. August 6, 2018; 150 (8): 1203-1213.
	Gabapentin Modulates HCN4 Channel Voltage-Dependence., Tae HS., Front Pharmacol. May 26, 2017; 8 554.
	Regulation of human cardiac potassium channels by full-length KCNE3 and KCNE4., Abbott GW., Sci Rep. December 6, 2016; 6 38412.
	Patch-clamp fluorometry-based channel counting to determine HCN channel conductance., Liu C., J Gen Physiol. July 1, 2016; 148 (1): 65-76.
	An N-terminal deletion variant of HCN1 in the epileptic WAG/Rij strain modulates HCN current densities., Wemhöner K., Front Mol Neurosci. November 3, 2015; 8 63.
	Binding of the auxiliary subunit TRIP8b to HCN channels shifts the mode of action of cAMP., Hu L., J Gen Physiol. December 1, 2013; 142 (6): 599-612.
	Asymmetric divergence in structure and function of HCN channel duplicates in Ciona intestinalis., Jackson HA., PLoS One. January 1, 2012; 7 (11): e47590.
	State-dependent accessibility of the P-S6 linker of pacemaker (HCN) channels supports a dynamic pore-to-gate coupling model., Siu CW., J Membr Biol. July 1, 2009; 230 (1): 35-47.
	Intracellular Mg2+ is a voltage-dependent pore blocker of HCN channels., Vemana S., Am J Physiol Cell Physiol. August 1, 2008; 295 (2): C557-65.
	Kinetic relationship between the voltage sensor and the activation gate in spHCN channels., Bruening-Wright A., J Gen Physiol. July 1, 2007; 130 (1): 71-81.
	Voltage sensor movement and cAMP binding allosterically regulate an inherently voltage-independent closed-open transition in HCN channels., Chen S., J Gen Physiol. February 1, 2007; 129 (2): 175-88.
	Mode shifts in the voltage gating of the mouse and human HCN2 and HCN4 channels., Elinder F., J Physiol. September 1, 2006; 575 (Pt 2): 417-31.
	Hysteresis in the voltage dependence of HCN channels: conversion between two modes affects pacemaker properties., Männikkö R., J Gen Physiol. March 1, 2005; 125 (3): 305-26.
	Salt bridges and gating in the COOH-terminal region of HCN2 and CNGA1 channels., Craven KB., J Gen Physiol. December 1, 2004; 124 (6): 663-77.
	Changes in local S4 environment provide a voltage-sensing mechanism for mammalian hyperpolarization-activated HCN channels., Bell DC., J Gen Physiol. January 1, 2004; 123 (1): 5-19.
	S4 movement in a mammalian HCN channel., Vemana S., J Gen Physiol. January 1, 2004; 123 (1): 21-32.
	KCNE2 modulates current amplitudes and activation kinetics of HCN4: influence of KCNE family members on HCN4 currents., Decher N., Pflugers Arch. September 1, 2003; 446 (6): 633-40.
	Regulation of hyperpolarization-activated HCN channel gating and cAMP modulation due to interactions of COOH terminus and core transmembrane regions., Wang J., J Gen Physiol. September 1, 2001; 118 (3): 237-50.
	Properties of hyperpolarization-activated pacemaker current defined by coassembly of HCN1 and HCN2 subunits and basal modulation by cyclic nucleotide., Chen S., J Gen Physiol. May 1, 2001; 117 (5): 491-504.

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