Papers associated with tail (and cdc42)

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	Caveolin 1 is required for axonal outgrowth of motor neurons and affects Xenopus neuromuscular development., Breuer M., Sci Rep. October 5, 2020; 10 (1): 16446.
	Glycogen synthase kinase 3 controls migration of the neural crest lineage in mouse and Xenopus., Gonzalez Malagon SG., Nat Commun. March 19, 2018; 9 (1): 1126.
	Control of actin polymerization via the coincidence of phosphoinositides and high membrane curvature., Daste F., J Cell Biol. November 6, 2017; 216 (11): 3745-3765.
	Id genes are essential for early heart formation., Cunningham TJ., Genes Dev. July 1, 2017; 31 (13): 1325-1338.
	The cellular and molecular mechanisms of tissue repair and regeneration as revealed by studies in Xenopus., Li J., Regeneration (Oxf). October 28, 2016; 3 (4): 198-208.
	Xenopus Rnd1 and Rnd3 GTP-binding proteins are expressed under the control of segmentation clock and required for somite formation., Goda T., Dev Dyn. November 1, 2009; 238 (11): 2867-76.
	Cadherin-11 regulates protrusive activity in Xenopus cranial neural crest cells upstream of Trio and the small GTPases., Kashef J., Genes Dev. June 15, 2009; 23 (12): 1393-8.
	Retinoic acid-inducible G protein-coupled receptors bind to frizzled receptors and may activate non-canonical Wnt signaling., Harada Y., Biochem Biophys Res Commun. July 13, 2007; 358 (4): 968-75.
	Defining synphenotype groups in Xenopus tropicalis by use of antisense morpholino oligonucleotides., Rana AA., PLoS Genet. November 17, 2006; 2 (11): e193.
	JNK and ROKalpha function in the noncanonical Wnt/RhoA signaling pathway to regulate Xenopus convergent extension movements., Kim GH., Dev Dyn. April 1, 2005; 232 (4): 958-68.
	Zebrafish Mir antagonizes Frizzled 7-induced gastrulation defects., Knowlton MN., Zebrafish. January 1, 2004; 1 (2): 133-44.
	Signal transduction. N-WASP regulation--the sting in the tail., Fawcett J., Science. October 27, 2000; 290 (5492): 725-6.
	The neuronal architecture of Xenopus retinal ganglion cells is sculpted by rho-family GTPases in vivo., Ruchhoeft ML., J Neurosci. October 1, 1999; 19 (19): 8454-63.
	Rho family GTPases control entry of Shigella flexneri into epithelial cells but not intracellular motility., Mounier J., J Cell Sci. July 1, 1999; 112 ( Pt 13) 2069-80.
	Neural Wiskott-Aldrich syndrome protein is implicated in the actin-based motility of Shigella flexneri., Suzuki T., EMBO J. May 15, 1998; 17 (10): 2767-76.

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