Papers associated with multicellular anatomical structure (and abcc8)

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	Binding of sulphonylureas to plasma proteins - A KATP channel perspective., Proks P., PLoS One. May 15, 2018; 13 (5): e0197634.
	Xenopus as a model system for studying pancreatic development and diabetes., Kofent J., Semin Cell Dev Biol. March 1, 2016; 51 106-16.
	Kir6.2 activation by sulfonylurea receptors: a different mechanism of action for SUR1 and SUR2A subunits via the same residues., Principalli MA., Physiol Rep. September 1, 2015; 3 (9):
	Recessive mutations in PCBD1 cause a new type of early-onset diabetes., Simaite D., Diabetes. October 1, 2014; 63 (10): 3557-64.
	The unusual stoichiometry of ADP activation of the KATP channel., Hosy E., Front Physiol. January 28, 2014; 5 11.
	Purinergic receptor-induced Ca2+ signaling in the neuroepithelium of the vomeronasal organ of larval Xenopus laevis., Dittrich K., Purinergic Signal. January 1, 2014; 10 (2): 327-36.
	Molecular mechanism of sulphonylurea block of K(ATP) channels carrying mutations that impair ATP inhibition and cause neonatal diabetes., Proks P., Diabetes. November 1, 2013; 62 (11): 3909-19.
	A universally conserved residue in the SUR1 subunit of the KATP channel is essential for translating nucleotide binding at SUR1 into channel opening., de Wet H., J Physiol. October 15, 2012; 590 (20): 5025-36.
	Homeoprotein hhex-induced conversion of intestinal to ventral pancreatic precursors results in the formation of giant pancreata in Xenopus embryos., Zhao H., Proc Natl Acad Sci U S A. May 29, 2012; 109 (22): 8594-9.
	Transient expression of Ngn3 in Xenopus endoderm promotes early and ectopic development of pancreatic beta and delta cells., Oropeza D., Genesis. March 1, 2012; 50 (3): 271-85.
	Engineering of an artificial light-modulated potassium channel., Caro LN., PLoS One. January 1, 2012; 7 (8): e43766.
	Neurally Derived Tissues in Xenopus laevis Embryos Exhibit a Consistent Bioelectrical Left-Right Asymmetry., Pai VP., Stem Cells Int. January 1, 2012; 2012 353491.
	The ATP-sensitive K(+)-channel (K(ATP)) controls early left-right patterning in Xenopus and chick embryos., Aw S., Dev Biol. October 1, 2010; 346 (1): 39-53.
	Activation of the K(ATP) channel by Mg-nucleotide interaction with SUR1., Proks P., J Gen Physiol. October 1, 2010; 136 (4): 389-405.
	Targeting hypertension with a new adenosine triphosphate-sensitive potassium channel opener iptakalim., Pan Z., J Cardiovasc Pharmacol. September 1, 2010; 56 (3): 215-28.
	Xenopus pancreas development., Pearl EJ., Dev Dyn. June 1, 2009; 238 (6): 1271-86.
	How ATP inhibits the open K(ATP) channel., Craig TJ., J Gen Physiol. July 1, 2008; 132 (1): 131-44.
	Expression of complement components coincides with early patterning and organogenesis in Xenopus laevis., McLin VA., Int J Dev Biol. January 1, 2008; 52 (8): 1123-33.
	Uncoupling by (--)-epigallocatechin-3-gallate of ATP-sensitive potassium channels from phosphatidylinositol polyphosphates and ATP., Jin JY., Pharmacol Res. September 1, 2007; 56 (3): 237-47.
	Remodelling of the SUR-Kir6.2 interface of the KATP channel upon ATP binding revealed by the conformational blocker rhodamine 123., Hosy E., J Physiol. July 1, 2007; 582 (Pt 1): 27-39.
	A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetes., Proks P., Hum Mol Genet. June 1, 2006; 15 (11): 1793-800.
	Functional effects of KCNJ11 mutations causing neonatal diabetes: enhanced activation by MgATP., Proks P., Hum Mol Genet. September 15, 2005; 14 (18): 2717-26.
	Ligand-dependent linkage of the ATP site to inhibition gate closure in the KATP channel., Li L., J Gen Physiol. September 1, 2005; 126 (3): 285-99.
	Effect of two amino acids in TM17 of Sulfonylurea receptor SUR1 on the binding of ATP-sensitive K+ channel modulators., Hambrock A., Diabetes. December 1, 2004; 53 Suppl 3 S128-34.
	Mapping the architecture of the ATP-binding site of the KATP channel subunit Kir6.2., Dabrowski M., J Physiol. June 1, 2004; 557 (Pt 2): 347-54.
	Potent and selective activation of the pancreatic beta-cell type K(ATP) channel by two novel diazoxide analogues., Dabrowski M., Diabetologia. October 1, 2003; 46 (10): 1375-82.
	The Mix family homeodomain gene bonnie and clyde functions with other components of the Nodal signaling pathway to regulate neural patterning in zebrafish., Trinh LA., Development. October 1, 2003; 130 (20): 4989-98.
	N-terminal transmembrane domain of the SUR controls trafficking and gating of Kir6 channel subunits., Chan KW., EMBO J. August 1, 2003; 22 (15): 3833-43.
	Differential selectivity of insulin secretagogues: mechanisms, clinical implications, and drug interactions., Gribble FM., J Diabetes Complications. January 1, 2003; 17 (2 Suppl): 11-5.
	K(ATP) channel activity is required for hatching in Xenopus embryos., Cheng SM., Dev Dyn. December 1, 2002; 225 (4): 588-91.
	Mutations in the linker domain of NBD2 of SUR inhibit transduction but not nucleotide binding., Matsuo M., EMBO J. August 15, 2002; 21 (16): 4250-8.
	The novel diazoxide analog 3-isopropylamino-7-methoxy-4H-1,2,4-benzothiadiazine 1,1-dioxide is a selective Kir6.2/SUR1 channel opener., Dabrowski M., Diabetes. June 1, 2002; 51 (6): 1896-906.
	Open state destabilization by ATP occupancy is mechanism speeding burst exit underlying KATP channel inhibition by ATP., Li L., J Gen Physiol. January 1, 2002; 119 (1): 105-16.
	Mutations within the P-loop of Kir6.2 modulate the intraburst kinetics of the ATP-sensitive potassium channel., Proks P., J Gen Physiol. October 1, 2001; 118 (4): 341-53.
	Structural basis for the interference between nicorandil and sulfonylurea action., Reimann F., Diabetes. October 1, 2001; 50 (10): 2253-9.
	Sulfonylurea receptors inhibit the epithelial sodium channel (ENaC) by reducing surface expression., Konstas AA., Pflugers Arch. August 1, 2001; 442 (5): 752-61.
	Effect of repaglinide on cloned beta cell, cardiac and smooth muscle types of ATP-sensitive potassium channels., Dabrowski M., Diabetologia. June 1, 2001; 44 (6): 747-56.
	Chromanol 293B, a blocker of the slow delayed rectifier K+ current (IKs), inhibits the CFTR Cl- current., Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. June 1, 2001; 363 (6): 590-6.
	Glimepiride block of cloned beta-cell, cardiac and smooth muscle K(ATP) channels., Song DK., Br J Pharmacol. May 1, 2001; 133 (1): 193-9.
	Effects of mitiglinide (S 21403) on Kir6.2/SUR1, Kir6.2/SUR2A and Kir6.2/SUR2B types of ATP-sensitive potassium channel., Reimann F., Br J Pharmacol. April 1, 2001; 132 (7): 1542-8.
	The antimalarial agent mefloquine inhibits ATP-sensitive K-channels., Gribble FM., Br J Pharmacol. October 1, 2000; 131 (4): 756-60.
	Fast1 is required for the development of dorsal axial structures in zebrafish., Sirotkin HI., Curr Biol. September 7, 2000; 10 (17): 1051-4.
	Nucleotide modulation of pinacidil stimulation of the cloned K(ATP) channel Kir6.2/SUR2A., Gribble FM., Mol Pharmacol. June 1, 2000; 57 (6): 1256-61.
	Molecular basis for K(ATP) assembly: transmembrane interactions mediate association of a K+ channel with an ABC transporter., Schwappach B., Neuron. April 1, 2000; 26 (1): 155-67.
	Rat homolog of sulfonylurea receptor 2B determines glibenclamide sensitivity of ROMK2 in Xenopus laevis oocyte., Tanemoto M., Am J Physiol Renal Physiol. April 1, 2000; 278 (4): F659-66.
	Altered functional properties of KATP channel conferred by a novel splice variant of SUR1., Sakura H., J Physiol. December 1, 1999; 521 Pt 2 337-50.
	Pharmacological plasticity of cardiac ATP-sensitive potassium channels toward diazoxide revealed by ADP., D'hahan N., Proc Natl Acad Sci U S A. October 12, 1999; 96 (21): 12162-7.
	A novel sulfonylurea receptor family member expressed in the embryonic Drosophila dorsal vessel and tracheal system., Nasonkin I., J Biol Chem. October 8, 1999; 274 (41): 29420-5.
	Effects of pond water, sediment, and sediment extracts from minnesota and vermont, USA, on early development and metamorphosis of xenopus., Fort DJ., Environ Toxicol Chem. October 1, 1999; 18 (10): 2305-2315.
	Interaction of vanadate with the cloned beta cell K(ATP) channel., Proks P., J Biol Chem. September 3, 1999; 274 (36): 25393-7.

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