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POLθ prevents MRE11- NBS1- CtIP-dependent fork breakage in the absence of BRCA2/RAD51 by filling lagging-strand gaps. , Mann A., Mol Cell. November 17, 2022; 82 (22): 4218-4231.e8.
RPA-coated single-stranded DNA promotes the ETAA1-dependent activation of ATR. , Lyu K., Cell Cycle. April 1, 2019; 18 (8): 898-913.
Fanconi-Anemia-Associated Mutations Destabilize RAD51 Filaments and Impair Replication Fork Protection. , Zadorozhny K., Cell Rep. October 10, 2017; 21 (2): 333-340.
Smarcal1-Mediated Fork Reversal Triggers Mre11-Dependent Degradation of Nascent DNA in the Absence of Brca2 and Stable Rad51 Nucleofilaments. , Kolinjivadi AM., Mol Cell. September 7, 2017; 67 (5): 867-881.e7.
Xenopus Mcm10 is a CDK-substrate required for replication fork stability. , Chadha GS., Cell Cycle. August 17, 2016; 15 (16): 2183-2195.
The Mre11- Rad50- Nbs1 (MRN) complex has a specific role in the activation of Chk1 in response to stalled replication forks. , Lee J ., Mol Biol Cell. May 1, 2013; 24 (9): 1343-53.
RAD51- and MRE11-dependent reassembly of uncoupled CMG helicase complex at collapsed replication forks. , Hashimoto Y., Nat Struct Mol Biol. December 4, 2011; 19 (1): 17-24.
Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair. , Peterson SE., J Cell Biol. September 5, 2011; 194 (5): 705-20.
Rad51 protects nascent DNA from Mre11-dependent degradation and promotes continuous DNA synthesis. , Hashimoto Y., Nat Struct Mol Biol. November 1, 2010; 17 (11): 1305-11.