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Summary Expression Phenotypes Gene Literature (182) GO Terms (6) Nucleotides (120) Proteins (54) Interactants (222) Wiki
XB-GENEPAGE-981216

Papers associated with nav1



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referenced by:

Developmental expression of the novel voltage-gated sodium channel auxiliary subunit beta3, in rat CNS., Shah BS, Stevens EB, Pinnock RD, Dixon AK, Lee K., J Physiol. August 1, 2001; 534 (Pt 3): 763-76.

Nav1.3 sodium channels: rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons., Cummins TR, Aglieco F, Renganathan M, Herzog RI, Dib-Hajj SD, Waxman SG., J Neurosci. August 15, 2001; 21 (16): 5952-61.

Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels., Vijayaragavan K, O'Leary ME, Chahine M., J Neurosci. October 15, 2001; 21 (20): 7909-18.

Point mutations in homology domain II modify the sensitivity of rat Nav1.8 sodium channels to the pyrethroid insecticide cismethrin., Soderlun DM, Lee SH., Neurotoxicology. December 1, 2001; 22 (6): 755-65.

Repressor element-1 silencing transcription/neuron-restrictive silencer factor is required for neural sodium channel expression during development of Xenopus., Armisén R, Fuentes R, Olguín P, Cabrejos ME, Kukuljan M., J Neurosci. October 1, 2002; 22 (19): 8347-51.                

Sodium channel heterologous expression in mammalian cells and the role of the endogenous beta1-subunits., Moran O, Conti F, Tammaro P., Neurosci Lett. January 23, 2003; 336 (3): 175-9.

Lidocaine block of neonatal Nav1.3 is differentially modulated by co-expression of beta1 and beta3 subunits., Lenkowski PW, Shah BS, Dinn AE, Lee K, Patel MK., Eur J Pharmacol. April 25, 2003; 467 (1-3): 23-30.

Importance of the conserved aromatic residues in the scorpion alpha-like toxin BmK M1: the hydrophobic surface region revisited., Sun YM, Bosmans F, Zhu RH, Goudet C, Xiong YM, Tytgat J, Wang DC., J Biol Chem. June 27, 2003; 278 (26): 24125-31.

Inhibition of cardiac sodium currents by toluene exposure., Cruz SL, Orta-Salazar G, Gauthereau MY, Millan-Perez Peña L, Salinas-Stefanón EM., Br J Pharmacol. October 1, 2003; 140 (4): 653-60.

Closing and inactivation potentiate the cocaethylene inhibition of cardiac sodium channels by distinct mechanisms., O'Leary ME, Digregorio M, Chahine M., Mol Pharmacol. December 1, 2003; 64 (6): 1575-85.

Molecular cloning and functional expression of the human sodium channel beta1B subunit, a novel splicing variant of the beta1 subunit., Qin N, D'Andrea MR, Lubin ML, Shafaee N, Codd EE, Correa AM., Eur J Biochem. December 1, 2003; 270 (23): 4762-70.

Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C., Vijayaragavan K, Boutjdir M, Chahine M., J Neurophysiol. April 1, 2004; 91 (4): 1556-69.

Differential modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by the local anesthetic lidocaine., Chevrier P, Vijayaragavan K, Chahine M., Br J Pharmacol. June 1, 2004; 142 (3): 576-84.

Role of auxiliary beta1-, beta2-, and beta3-subunits and their interaction with Na(v)1.8 voltage-gated sodium channel., Vijayaragavan K, Powell AJ, Kinghorn IJ, Chahine M., Biochem Biophys Res Commun. June 25, 2004; 319 (2): 531-40.

Occurrence of a tetrodotoxin-sensitive calcium current in rat ventricular myocytes after long-term myocardial infarction., Alvarez JL, Salinas-Stefanon E, Orta G, Ferrer T, Talavera K, Galán L, Vassort G., Cardiovasc Res. September 1, 2004; 63 (4): 653-61.

Cyclosporin A and deltamethrin block the downregulation of Nav1.8 sodium channels expressed in Xenopus oocytes., Choi JS, Soderlund DM., Neurosci Lett. September 9, 2004; 367 (3): 389-93.

State-dependent trapping of flecainide in the cardiac sodium channel., Ramos E, O'leary ME., J Physiol. October 1, 2004; 560 (Pt 1): 37-49.

The poison Dart frog's batrachotoxin modulates Nav1.8., Bosmans F, Maertens C, Verdonck F, Tytgat J., FEBS Lett. November 5, 2004; 577 (1-2): 245-8.

Use-dependent potentiation of the Nav1.6 sodium channel., Zhou W, Goldin AL., Biophys J. December 1, 2004; 87 (6): 3862-72.

A role for the carbohydrate portion of ginsenoside Rg3 in Na+ channel inhibition., Kim JH, Hong YH, Lee JH, Lee JH, Kim DH, Nam G, Jeong SM, Lee BH, Lee SM, Nah SY., Mol Cells. February 28, 2005; 19 (1): 137-42.

Molecular basis of the mammalian potency of the scorpion alpha-like toxin, BmK M1., Liu LH, Bosmans F, Maertens C, Zhu RH, Wang DC, Tytgat J., FASEB J. April 1, 2005; 19 (6): 594-6.

State-dependent block of rat Nav1.4 sodium channels expressed in xenopus oocytes by pyrazoline-type insecticides., Silver K, Soderlund DM., Neurotoxicology. June 1, 2005; 26 (3): 397-406.

A new type of scorpion Na+-channel-toxin-like polypeptide active on K+ channels., Srairi-Abid N, Guijarro JI, Benkhalifa R, Mantegazza M, Cheikh A, Ben Aissa M, Haumont PY, Delepierre M, El Ayeb M., Biochem J. June 1, 2005; 388 (Pt 2): 455-64.

Distribution and functional characterization of human Nav1.3 splice variants., Thimmapaya R, Neelands T, Niforatos W, Davis-Taber RA, Choi W, Putman CB, Kroeger PE, Packer J, Gopalakrishnan M, Faltynek CR, Surowy CS, Scott VE., Eur J Neurosci. July 1, 2005; 22 (1): 1-9.

Structural basis for the voltage-gated Na+ channel selectivity of the scorpion alpha-like toxin BmK M1., Ye X, Bosmans F, Li C, Zhang Y, Wang DC, Tytgat J., J Mol Biol. November 4, 2005; 353 (4): 788-803.

A mutation in the local anaesthetic binding site abolishes toluene effects in sodium channels., Gauthereau MY, Salinas-Stefanon EM, Cruz SL., Eur J Pharmacol. December 28, 2005; 528 (1-3): 17-26.

RE-1 silencer of transcription/neural restrictive silencer factor modulates ectodermal patterning during Xenopus development., Olguín P, Oteíza P, Gamboa E, Gómez-Skármeta JL, Kukuljan M., J Neurosci. March 8, 2006; 26 (10): 2820-9.                    

An epilepsy mutation in the sodium channel SCN1A that decreases channel excitability., Barela AJ, Waddy SP, Lickfett JG, Hunter J, Anido A, Helmers SL, Goldin AL, Escayg A., J Neurosci. March 8, 2006; 26 (10): 2714-23.

GLUT8 is dispensable for embryonic development but influences hippocampal neurogenesis and heart function., Membrez M, Hummler E, Beermann F, Haefliger JA, Savioz R, Pedrazzini T, Thorens B., Mol Cell Biol. June 1, 2006; 26 (11): 4268-76.

Potent modulation of the voltage-gated sodium channel Nav1.7 by OD1, a toxin from the scorpion Odonthobuthus doriae., Maertens C, Cuypers E, Amininasab M, Jalali A, Vatanpour H, Tytgat J., Mol Pharmacol. July 1, 2006; 70 (1): 405-14.

Augmentation of Cav3.2 T-type calcium channel activity by cAMP-dependent protein kinase A., Kim JA, Park JY, Kang HW, Huh SU, Jeong SW, Lee JH, Lee JH., J Pharmacol Exp Ther. July 1, 2006; 318 (1): 230-7.

muO-conotoxin MrVIB selectively blocks Nav1.8 sensory neuron specific sodium channels and chronic pain behavior without motor deficits., Ekberg J, Jayamanne A, Vaughan CW, Aslan S, Thomas L, Mould J, Drinkwater R, Baker MD, Abrahamsen B, Wood JN, Adams DJ, Christie MJ, Lewis RJ., Proc Natl Acad Sci U S A. November 7, 2006; 103 (45): 17030-5.

Isolation and structure-activity of mu-conotoxin TIIIA, a potent inhibitor of tetrodotoxin-sensitive voltage-gated sodium channels., Lewis RJ, Schroeder CI, Ekberg J, Nielsen KJ, Loughnan M, Thomas L, Adams DA, Drinkwater R, Adams DJ, Alewood PF., Mol Pharmacol. March 1, 2007; 71 (3): 676-85.

A cation-pi interaction discriminates among sodium channels that are either sensitive or resistant to tetrodotoxin block., Santarelli VP, Eastwood AL, Dougherty DA, Horn R, Ahern CA., J Biol Chem. March 16, 2007; 282 (11): 8044-51.

H+ pump-dependent changes in membrane voltage are an early mechanism necessary and sufficient to induce Xenopus tail regeneration., Adams DS, Masi A, Levin M., Development. April 1, 2007; 134 (7): 1323-35.          

Modulatory effect of auxiliary beta1 subunit on Nav1.3 voltage-gated sodium channel expressed in Xenopus oocyte., Wang YW, Cheng ZJ, Tan H, Xia YM, Ren RR, Ding YQ., Chin Med J (Engl). April 20, 2007; 120 (8): 721-3.

Effects of dextrorotatory morphinans on brain Na+ channels expressed in Xenopus oocytes., Lee JH, Lee JH, Shin EJ, Jeong SM, Lee BH, Yoon IS, Lee JH, Lee JH, Choi SH, Kim YH, Pyo MK, Lee SM, Chae JS, Rhim H, Oh JW, Kim HC, Nah SY., Eur J Pharmacol. June 14, 2007; 564 (1-3): 7-17.

A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore., Struyk AF, Cannon SC., J Gen Physiol. July 1, 2007; 130 (1): 11-20.          

Solution structure of Jingzhaotoxin-III, a peptide toxin inhibiting both Nav1.5 and Kv2.1 channels., Liao Z, Yuan C, Peng K, Xiao Y, Liang S., Toxicon. July 1, 2007; 50 (1): 135-43.

Nav channel mechanosensitivity: activation and inactivation accelerate reversibly with stretch., Morris CE, Juranka PF., Biophys J. August 1, 2007; 93 (3): 822-33.

Structure and sodium channel activity of an excitatory I1-superfamily conotoxin., Buczek O, Wei D, Babon JJ, Yang X, Fiedler B, Chen P, Yoshikami D, Olivera BM, Bulaj G, Norton RS., Biochemistry. September 4, 2007; 46 (35): 9929-40.

Concentration-dependent accumulation of [3H]-deltamethrin in sodium channel Nav1.2/beta1 expressing Xenopus laevis oocytes., Watkins JA, Meacham CA, Crofton KM, Shafer TJ., Toxicol In Vitro. December 1, 2007; 21 (8): 1672-7.

Combination of cardiac conduction disease and long QT syndrome caused by mutation T1620K in the cardiac sodium channel., Surber R, Hensellek S, Prochnau D, Werner GS, Benndorf K, Figulla HR, Zimmer T., Cardiovasc Res. March 1, 2008; 77 (4): 740-8.

Modifications of aliphatic side chain of 20(S)-ginsenoside RG3 cause an enhancement or loss of brain Na+ channel current inhibitions., Lee JH, Lee JH, Choi SH, Lee BH, Yoon IS, Shin TJ, Pyo MK, Lee SM, Rhim H, Park MH, Park TY, Nah SY., Biol Pharm Bull. March 1, 2008; 31 (3): 480-6.

Effects of BmK AS on Nav1.2 expressed in Xenopus laevis oocytes., Tan M, Zhu MM, Liu Y, Cheng HW, Ji YH., Cell Biol Toxicol. April 1, 2008; 24 (2): 143-9.

Involvement of batrachotoxin binding sites in ginsenoside-mediated voltage-gated Na+ channel regulation., Lee JH, Lee BH, Choi SH, Yoon IS, Shin TJ, Pyo MK, Lee SM, Kim HC, Nah SY., Brain Res. April 8, 2008; 1203 61-7.

An extracellular Cu2+ binding site in the voltage sensor of BK and Shaker potassium channels., Ma Z, Wong KY, Horrigan FT., J Gen Physiol. May 1, 2008; 131 (5): 483-502.                      

Alpha-scorpion toxin impairs a conformational change that leads to fast inactivation of muscle sodium channels., Campos FV, Chanda B, Beirão PS, Bezanilla F., J Gen Physiol. August 1, 2008; 132 (2): 251-63.                      

Correlations between clinical and physiological consequences of the novel mutation R878C in a highly conserved pore residue in the cardiac Na+ channel., Zhang Y, Wang T, Ma A, Zhou X, Gui J, Wan H, Shi R, Huang C, Grace AA, Huang CL, Trump D, Zhang H, Zimmer T, Lei M., Acta Physiol (Oxf). December 1, 2008; 194 (4): 311-23.            

Human and rat Nav1.3 voltage-gated sodium channels differ in inactivation properties and sensitivity to the pyrethroid insecticide tefluthrin., Tan J, Soderlund DM., Neurotoxicology. January 1, 2009; 30 (1): 81-9.

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