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Summary Expression Phenotypes Gene Literature (182) GO Terms (6) Nucleotides (120) Proteins (54) Interactants (222) Wiki
XB-GENEPAGE-981216

Papers associated with nav1



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A novel µ-conopeptide, CnIIIC, exerts potent and preferential inhibition of NaV1.2/1.4 channels and blocks neuronal nicotinic acetylcholine receptors., Favreau P, Benoit E, Hocking HG, Carlier L, D' hoedt D, Leipold E, Markgraf R, Schlumberger S, Córdova MA, Gaertner H, Paolini-Bertrand M, Hartley O, Tytgat J, Heinemann SH, Bertrand D, Boelens R, Stöcklin R, Molgó J., Br J Pharmacol. July 1, 2012; 166 (5): 1654-68.

Differential state-dependent modification of inactivation-deficient Nav1.6 sodium channels by the pyrethroid insecticides S-bioallethrin, tefluthrin and deltamethrin., McCavera SJ, Soderlund DM., Neurotoxicology. June 1, 2012; 33 (3): 384-90.

Pharmacological kinetics of BmK AS, a sodium channel site 4-specific modulator on Nav1.3., Liu ZR, Tao J, Dong BQ, Ding G, Cheng ZJ, He HQ, Ji YH., Neurosci Bull. June 1, 2012; 28 (3): 209-21.

18β-Glycyrrhetinic acid preferentially blocks late Na current generated by ΔKPQ Nav1.5 channels., Du YM, Xia CK, Zhao N, Dong Q, Lei M, Xia JH., Acta Pharmacol Sin. June 1, 2012; 33 (6): 752-60.            

Infrared light excites cells by changing their electrical capacitance., Shapiro MG, Homma K, Villarreal S, Richter CP, Bezanilla F., Nat Commun. March 13, 2012; 3 736.          

JZTX-XIII, a Kv channel gating modifier toxin from Chinese tarantula Chilobrachys jingzhao., Yuan C, Liu Z, Hu W, Gao T, Liang S., Toxicon. February 1, 2012; 59 (2): 265-71.

Transmembrane voltage potential controls embryonic eye patterning in Xenopus laevis., Pai VP, Aw S, Shomrat T, Lemire JM, Levin M., Development. January 1, 2012; 139 (2): 313-23.                

Estimating the voltage-dependent free energy change of ion channels using the median voltage for activation., Chowdhury S, Chanda B., J Gen Physiol. January 1, 2012; 139 (1): 3-17.            

A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype., Gosselin-Badaroudine P, Keller DI, Huang H, Pouliot V, Chatelier A, Osswald S, Brink M, Chahine M., PLoS One. January 1, 2012; 7 (5): e38331.          

Extracellular proton modulation of the cardiac voltage-gated sodium channel, Nav1.5., Jones DK, Peters CH, Tolhurst SA, Claydon TW, Ruben PC., Biophys J. November 2, 2011; 101 (9): 2147-56.

Navβ subunits modulate the inhibition of Nav1.8 by the analgesic gating modifier μO-conotoxin MrVIB., Wilson MJ, Zhang MM, Azam L, Olivera BM, Bulaj G, Yoshikami D., J Pharmacol Exp Ther. August 1, 2011; 338 (2): 687-93.

Functional properties and toxin pharmacology of a dorsal root ganglion sodium channel viewed through its voltage sensors., Bosmans F, Puopolo M, Martin-Eauclaire MF, Bean BP, Swartz KJ., J Gen Physiol. July 1, 2011; 138 (1): 59-72.                  

μ-Conotoxins that differentially block sodium channels NaV1.1 through 1.8 identify those responsible for action potentials in sciatic nerve., Wilson MJ, Yoshikami D, Azam L, Gajewiak J, Olivera BM, Bulaj G, Zhang MM., Proc Natl Acad Sci U S A. June 21, 2011; 108 (25): 10302-7.

Expression of the voltage- and Ca2+-dependent BK potassium channel subunits BKβ1 and BKβ4 in rodent astrocytes., Seidel KN, Derst C, Salzmann M, Höltje M, Priller J, Markgraf R, Heinemann SH, Heilmann H, Skatchkov SN, Eaton MJ, Veh RW, Prüss H., Glia. June 1, 2011; 59 (6): 893-902.

Independent and joint modulation of rat Nav1.6 voltage-gated sodium channels by coexpression with the auxiliary β1 and β2 subunits., Tan J, Soderlund DM., Biochem Biophys Res Commun. April 22, 2011; 407 (4): 788-92.

Localization of receptor site on insect sodium channel for depressant β-toxin BmK IT2., He H, Liu Z, Dong B, Zhang J, Shu X, Zhou J, Ji Y., PLoS One. January 7, 2011; 6 (1): e14510.            

Phyla- and Subtype-Selectivity of CgNa, a Na Channel Toxin from the Venom of the Giant Caribbean Sea Anemone Condylactis Gigantea., Billen B, Debaveye S, Béress L, Garateix A, Tytgat J., Front Pharmacol. November 23, 2010; 1 133.        

Molecular mechanism of allosteric modification of voltage-dependent sodium channels by local anesthetics., Arcisio-Miranda M, Muroi Y, Chowdhury S, Chanda B., J Gen Physiol. November 1, 2010; 136 (5): 541-54.                

Molecular determination of selectivity of the site 3 modulator (BmK I) to sodium channels in the CNS: a clue to the importance of Nav1.6 in BmK I-induced neuronal hyperexcitability., He H, Liu Z, Dong B, Zhou J, Zhu H, Ji Y., Biochem J. October 15, 2010; 431 (2): 289-98.

Induction of vertebrate regeneration by a transient sodium current., Tseng AS, Beane WS, Lemire JM, Masi A, Levin M., J Neurosci. September 29, 2010; 30 (39): 13192-200.                    

Pharmacological modulation of brain Nav1.2 and cardiac Nav1.5 subtypes by the local anesthetic ropivacaine., Cheng HW, Yang HT, Zhou JJ, Ji YH, Zhu HY., Neurosci Bull. August 1, 2010; 26 (4): 289-96.

Multiple loss-of-function mechanisms contribute to SCN5A-related familial sick sinus syndrome., Gui J, Wang T, Jones RP, Trump D, Zimmer T, Lei M., PLoS One. June 7, 2010; 5 (6): e10985.              

Optogenetic localization and genetic perturbation of saccade-generating neurons in zebrafish., Schoonheim PJ, Arrenberg AB, Del Bene F, Baier H., J Neurosci. May 19, 2010; 30 (20): 7111-20.

An in vivo tethered toxin approach for the cell-autonomous inactivation of voltage-gated sodium channel currents in nociceptors., Stürzebecher AS, Hu J, Smith ES, Frahm S, Santos-Torres J, Kampfrath B, Auer S, Lewin GR, Ibañez-Tallon I., J Physiol. May 15, 2010; 588 (Pt 10): 1695-707.        

Blocking effect of methylflavonolamine on human Na(V)1.5 channels expressed in Xenopus laevis oocytes and on sodium currents in rabbit ventricular myocytes., Fan XR, Ma JH, Zhang PH, Xing JL., Acta Pharmacol Sin. March 1, 2010; 31 (3): 297-306.                  

Electric currents in Xenopus tadpole tail regeneration., Reid B, Song B, Zhao M., Dev Biol. November 1, 2009; 335 (1): 198-207.                

Membrane trauma and Na+ leak from Nav1.6 channels., Wang JA, Lin W, Morris T, Banderali U, Juranka PF, Morris CE., Am J Physiol Cell Physiol. October 1, 2009; 297 (4): C823-34.

The external pore loop interacts with S6 and S3-S4 linker in domain 4 to assume an essential role in gating control and anticonvulsant action in the Na(+) channel., Yang YC, Hsieh JY, Kuo CC., J Gen Physiol. August 1, 2009; 134 (2): 95-113.                      

Slow inactivation of the NaV1.4 sodium channel in mammalian cells is impeded by co-expression of the beta1 subunit., Webb J, Wu FF, Cannon SC., Pflugers Arch. April 1, 2009; 457 (6): 1253-63.

Human and rat Nav1.3 voltage-gated sodium channels differ in inactivation properties and sensitivity to the pyrethroid insecticide tefluthrin., Tan J, Soderlund DM., Neurotoxicology. January 1, 2009; 30 (1): 81-9.

Synergistic and antagonistic interactions between tetrodotoxin and mu-conotoxin in blocking voltage-gated sodium channels., Zhang MM, McArthur JR, Azam L, Bulaj G, Olivera BM, French RJ, Yoshikami D., Channels (Austin). January 1, 2009; 3 (1): 32-8.

Correlations between clinical and physiological consequences of the novel mutation R878C in a highly conserved pore residue in the cardiac Na+ channel., Zhang Y, Wang T, Ma A, Zhou X, Gui J, Wan H, Shi R, Huang C, Grace AA, Huang CL, Trump D, Zhang H, Zimmer T, Lei M., Acta Physiol (Oxf). December 1, 2008; 194 (4): 311-23.            

Alpha-scorpion toxin impairs a conformational change that leads to fast inactivation of muscle sodium channels., Campos FV, Chanda B, Beirão PS, Bezanilla F., J Gen Physiol. August 1, 2008; 132 (2): 251-63.                      

An extracellular Cu2+ binding site in the voltage sensor of BK and Shaker potassium channels., Ma Z, Wong KY, Horrigan FT., J Gen Physiol. May 1, 2008; 131 (5): 483-502.                      

Involvement of batrachotoxin binding sites in ginsenoside-mediated voltage-gated Na+ channel regulation., Lee JH, Lee BH, Choi SH, Yoon IS, Shin TJ, Pyo MK, Lee SM, Kim HC, Nah SY., Brain Res. April 8, 2008; 1203 61-7.

Effects of BmK AS on Nav1.2 expressed in Xenopus laevis oocytes., Tan M, Zhu MM, Liu Y, Cheng HW, Ji YH., Cell Biol Toxicol. April 1, 2008; 24 (2): 143-9.

Combination of cardiac conduction disease and long QT syndrome caused by mutation T1620K in the cardiac sodium channel., Surber R, Hensellek S, Prochnau D, Werner GS, Benndorf K, Figulla HR, Zimmer T., Cardiovasc Res. March 1, 2008; 77 (4): 740-8.

Modifications of aliphatic side chain of 20(S)-ginsenoside RG3 cause an enhancement or loss of brain Na+ channel current inhibitions., Lee JH, Lee JH, Choi SH, Lee BH, Yoon IS, Shin TJ, Pyo MK, Lee SM, Rhim H, Park MH, Park TY, Nah SY., Biol Pharm Bull. March 1, 2008; 31 (3): 480-6.

Concentration-dependent accumulation of [3H]-deltamethrin in sodium channel Nav1.2/beta1 expressing Xenopus laevis oocytes., Watkins JA, Meacham CA, Crofton KM, Shafer TJ., Toxicol In Vitro. December 1, 2007; 21 (8): 1672-7.

Structure and sodium channel activity of an excitatory I1-superfamily conotoxin., Buczek O, Wei D, Babon JJ, Yang X, Fiedler B, Chen P, Yoshikami D, Olivera BM, Bulaj G, Norton RS., Biochemistry. September 4, 2007; 46 (35): 9929-40.

Nav channel mechanosensitivity: activation and inactivation accelerate reversibly with stretch., Morris CE, Juranka PF., Biophys J. August 1, 2007; 93 (3): 822-33.

A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore., Struyk AF, Cannon SC., J Gen Physiol. July 1, 2007; 130 (1): 11-20.          

Solution structure of Jingzhaotoxin-III, a peptide toxin inhibiting both Nav1.5 and Kv2.1 channels., Liao Z, Yuan C, Peng K, Xiao Y, Liang S., Toxicon. July 1, 2007; 50 (1): 135-43.

Effects of dextrorotatory morphinans on brain Na+ channels expressed in Xenopus oocytes., Lee JH, Lee JH, Shin EJ, Jeong SM, Lee BH, Yoon IS, Lee JH, Lee JH, Choi SH, Kim YH, Pyo MK, Lee SM, Chae JS, Rhim H, Oh JW, Kim HC, Nah SY., Eur J Pharmacol. June 14, 2007; 564 (1-3): 7-17.

Modulatory effect of auxiliary beta1 subunit on Nav1.3 voltage-gated sodium channel expressed in Xenopus oocyte., Wang YW, Cheng ZJ, Tan H, Xia YM, Ren RR, Ding YQ., Chin Med J (Engl). April 20, 2007; 120 (8): 721-3.

H+ pump-dependent changes in membrane voltage are an early mechanism necessary and sufficient to induce Xenopus tail regeneration., Adams DS, Masi A, Levin M., Development. April 1, 2007; 134 (7): 1323-35.          

A cation-pi interaction discriminates among sodium channels that are either sensitive or resistant to tetrodotoxin block., Santarelli VP, Eastwood AL, Dougherty DA, Horn R, Ahern CA., J Biol Chem. March 16, 2007; 282 (11): 8044-51.

Isolation and structure-activity of mu-conotoxin TIIIA, a potent inhibitor of tetrodotoxin-sensitive voltage-gated sodium channels., Lewis RJ, Schroeder CI, Ekberg J, Nielsen KJ, Loughnan M, Thomas L, Adams DA, Drinkwater R, Adams DJ, Alewood PF., Mol Pharmacol. March 1, 2007; 71 (3): 676-85.

muO-conotoxin MrVIB selectively blocks Nav1.8 sensory neuron specific sodium channels and chronic pain behavior without motor deficits., Ekberg J, Jayamanne A, Vaughan CW, Aslan S, Thomas L, Mould J, Drinkwater R, Baker MD, Abrahamsen B, Wood JN, Adams DJ, Christie MJ, Lewis RJ., Proc Natl Acad Sci U S A. November 7, 2006; 103 (45): 17030-5.

Potent modulation of the voltage-gated sodium channel Nav1.7 by OD1, a toxin from the scorpion Odonthobuthus doriae., Maertens C, Cuypers E, Amininasab M, Jalali A, Vatanpour H, Tytgat J., Mol Pharmacol. July 1, 2006; 70 (1): 405-14.

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